Agency
National Institute of Health (NIH)
Institute
National Institute of Neurological Disorders and Stroke (NINDS)
Type
Clinical Investigator Award (CIA) (K08)
Project #
5K08NS001530-05
Application #
2259427
Study Section
NST-2 Subcommittee (NST)
Project Start
1991-08-01
Project End
1997-06-30
Budget Start
1995-07-01
Budget End
1997-06-30
Support Year
5
Fiscal Year
1995
Total Cost
Indirect Cost
Name
University of Texas Health Science Center Houston
Department
Neurology
Type
Schools of Medicine
DUNS #
City
Houston
State
TX
Country
United States
Zip Code
77225
Vriesendorp, F J; Flynn, R E; Malone, M R et al. (1998) Systemic complement depletion reduces inflammation and demyelination in adoptive transfer experimental allergic neuritis. Acta Neuropathol (Berl) 95:297-301
Vriesendorp, F J (1997) Insights into Campylobacter jejuni-induced Guillain-Barre syndrome from the Lewis rat model of experimental allergic neuritis. J Infect Dis 176 Suppl 2:S164-8
Vriesendorp, F J; Quadri, S M; Flynn, R E et al. (1997) Preclinical analysis of radiolabeled anti-GD2 immunoglobulin G. Cancer 80:2642-9
Vriesendorp, F J; Flynn, R E; Khan, M et al. (1996) Oral administration of type I interferon modulates the course of experimental allergic neuritis. Autoimmunity 24:157-65
Vriesendorp, F J; Triggs, W J; Mayer, R F et al. (1995) Electrophysiological studies in Guillain-Barre syndrome: correlation with antibodies to GM1, GD1B and Campylobacter jejuni. J Neurol 242:460-5
Vriesendorp, F J; Flynn, R E; Pappolla, M A et al. (1995) Complement depletion affects demyelination and inflammation in experimental allergic neuritis. J Neuroimmunol 58:157-65
Vriesendorp, F J; Mishu, B; Blaser, M J et al. (1993) Serum antibodies to GM1, GD1b, peripheral nerve myelin, and Campylobacter jejuni in patients with Guillain-Barre syndrome and controls: correlation and prognosis. Ann Neurol 34:130-5