Molecular Basis for Variation of Influenza Viruses

Moscona, Anne

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Allergy and Infectious Diseases (NIAID)
• Type: Physician Scientist Award (K11)
• Project #: 5K11AI000739-03
• Application #: 3085115
• Study Section: Microbiology and Infectious Diseases Research Committee (MID)
• Project Start: 1987-04-01
• Project End: 1992-03-30
• Budget Start: 1989-04-01
• Budget End: 1990-03-31
• Support Year: 3
• Fiscal Year: 1989

Institution

• Name: Mount Sinai School of Medicine
• Type: Schools of Medicine
• City: New York
• State: NY
• Country: United States
• Zip Code: 10029

Publications

Sinha, Rajesh K; Park, Chung; Hwang, Il-Young et al. (2009) B lymphocytes exit lymph nodes through cortical lymphatic sinusoids by a mechanism independent of sphingosine-1-phosphate-mediated chemotaxis. Immunity 30:434-46

Moscona, A; Peluso, R W (1993) Persistent infection with human parainfluenza virus 3 in CV-1 cells: analysis of the role of defective interfering particles. Virology 194:399-402

Moscona, A; Peluso, R W (1992) Fusion properties of cells infected with human parainfluenza virus type 3: receptor requirements for viral spread and virus-mediated membrane fusion. J Virol 66:6280-7

Moscona, A; Peluso, R W (1991) Properties of human parainfluenza virus type 3 RNA polymerase/replicase activity in vitro: consensus with other negative-stranded RNA viruses. J Virol 65:4470-4

Moscona, A (1991) Defective interfering particles of human parainfluenza virus type 3 are associated with persistent infection in cell culture. Virology 183:821-4

Moscona, A; Peluso, R W (1991) Fusion properties of cells persistently infected with human parainfluenza virus type 3: participation of hemagglutinin-neuraminidase in membrane fusion. J Virol 65:2773-7

Moscona, A; Galinski, M S (1990) Characterization of human parainfluenza virus type 3 persistent infection in cell culture. J Virol 64:3212-8