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Guidot, D M; Kitlowski, A D; Hybertson, B M et al. (1999) Mitochondrial antioxidant function is a potential mechanism for organ differences in interleukin-1-mediated tolerance to oxidative injury. Am J Med Sci 318:308-15
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Guidot, D M (1998) Endotoxin pretreatment in vivo increases the mitochondrial respiratory capacity in rat hepatocytes. Arch Biochem Biophys 354:9-17
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Holguin, F; Moss, I; Brown, L A et al. (1998) Chronic ethanol ingestion impairs alveolar type II cell glutathione homeostasis and function and predisposes to endotoxin-mediated acute edematous lung injury in rats. J Clin Invest 101:761-8
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Guidot, D M (1996) Endotoxin treatment increases lung mitochondrial scavenging of extramitochondrial superoxide in hyperoxia-exposed rats. Arch Biochem Biophys 326:266-70
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Guidot, D M; Hybertson, B M; Kitlowski, R P et al. (1996) Inhaled NO prevents IL-1-induced neutrophil accumulation and associated acute edema in isolated rat lungs. Am J Physiol 271:L225-9
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Guidot, D M; Repine, J E; Kitlowski, A D et al. (1995) Mitochondrial respiration scavenges extramitochondrial superoxide anion via a nonenzymatic mechanism. J Clin Invest 96:1131-6
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Guidot, D M; Repine, M J; Hybertson, B M et al. (1995) Inhaled nitric oxide prevents neutrophil-mediated, oxygen radical-dependent leak in isolated rat lungs. Am J Physiol 269:L2-5
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Guidot, D M; Stevens, E E; Repine, M J et al. (1994) Intratracheal but not intravascular interleukin-1 causes acute edematous injury in isolated neutrophil-perfused rat lungs through an oxygen radical-mediated mechanism. J Lab Clin Med 123:605-9
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Guidot, D M; Repine, M J; Westcott, J Y et al. (1994) Intrinsic 5-lipoxygenase activity is required for neutrophil responsivity. Proc Natl Acad Sci U S A 91:8156-9
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Guidot, D M; Linas, S L; Repine, M J et al. (1994) Interleukin-1 treatment increases neutrophils but not antioxidant enzyme activity or resistance to ischemia-reperfusion injury in rat kidneys. Inflammation 18:537-45
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