Agency
National Institute of Health (NIH)
Institute
National Institute on Alcohol Abuse and Alcoholism (NIAAA)
Type
Research Project (R01)
Project #
5R01AA009115-04
Application #
2045322
Study Section
Biochemistry, Physiology and Medicine Subcommittee (ALCB)
Project Start
1992-08-01
Project End
1997-07-31
Budget Start
1995-08-01
Budget End
1997-07-31
Support Year
4
Fiscal Year
1995
Total Cost
Indirect Cost
Name
University of Florida
Department
Neurosciences
Type
Schools of Medicine
DUNS #
073130411
City
Gainesville
State
FL
Country
United States
Zip Code
32611
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Peris, J; Jung, B J; Resnick, A et al. (1998) Antisense inhibition of striatal GABAA receptor proteins decreases GABA-stimulated chloride uptake and increases cocaine sensitivity in rats. Brain Res Mol Brain Res 57:310-20
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Peris, J; Anderson, K J; Vickroy, T W et al. (1997) Neurochemical basis of disruption of hippocampal long term potentiation by chronic alcohol exposure. Front Biosci 2:d309-16
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Brazeau, G A; al-Suwayeh, S; Peris, J et al. (1995) Creatine kinase release from isolated EDL muscles in chronic ethanol-treated rats. Alcohol 12:145-9
Thinschmidt, J S; Kinney, G G; Kocsis, B (1995) The supramammillary nucleus: is it necessary for the mediation of hippocampal theta rhythm? Neuroscience 67:301-12
Tremwel, M F; Hunter, B E; Peris, J (1994) Chronic ethanol exposure enhances [3H]GABA release and does not affect GABAA receptor mediated 36Cl uptake. Synapse 17:149-54

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