Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Project (R01)
Project #
3R01AI025769-06S1
Application #
3139349
Study Section
Special Emphasis Panel (ARR (V1))
Project Start
1987-09-30
Project End
1993-07-31
Budget Start
1992-09-01
Budget End
1993-07-31
Support Year
6
Fiscal Year
1992
Total Cost
Indirect Cost

  Institution

Name
California Pacific Medical Center Research Institute
Department
Type
DUNS #
City
San Francisco
State
CA
Country
United States
Zip Code
94107

  Publications

Parker, A E; Bermudez, L E (2000) Sequence and characterization of the glyceraldehyde-3-phosphate dehydrogenase of Mycobacterium avium: correlation with an epidermal growth factor binding protein. Microb Pathog 28:135-44
Bermudez, L E; Parker, A; Petrofsky, M (1999) Apoptosis of Mycobacterium avium-infected macrophages is mediated by both tumour necrosis factor (TNF) and Fas, and involves the activation of caspases. Clin Exp Immunol 116:94-9
Bermudez, L E; Wu, M; Miltner, E et al. (1999) Isolation of two subpopulations of Mycobacterium avium within human macrophages. FEMS Microbiol Lett 178:19-26
Kim, S Y; Goodman, J R; Petrofsky, M et al. (1998) Mycobacterium avium infection of gut mucosa in mice associated with late inflammatory response and intestinal cell necrosis. J Med Microbiol 47:725-31
Mohagheghpour, N; Gammon, D; van Vollenhoven, A et al. (1997) Mycobacterium avium reduces expression of costimulatory/adhesion molecules by human monocytes. Cell Immunol 176:82-91
Parker, A E; Bermudez, L E (1997) Expression of the green fluorescent protein (GFP) in mycobacterium avium as a tool to study the interaction between Mycobacteria and host cells. Microb Pathog 22:193-8
Azouaou, N; Petrofsky, M; Young, L S et al. (1997) Mycobacterium avium infection in mice is associated with time-related expression of Th1 and Th2 CD4+ T-lymphocyte response. Immunology 91:414-20
Cirillo, J D; Falkow, S; Tompkins, L S et al. (1997) Interaction of Mycobacterium avium with environmental amoebae enhances virulence. Infect Immun 65:3759-67
Bermudez, L E; Parker, A; Goodman, J R (1997) Growth within macrophages increases the efficiency of Mycobacterium avium in invading other macrophages by a complement receptor-independent pathway. Infect Immun 65:1916-25
Bermudez, L E; Petrofsky, M; Shelton, K (1996) Epidermal growth factor-binding protein in Mycobacterium avium and Mycobacterium tuberculosis: a possible role in the mechanism of infection. Infect Immun 64:2917-22

Showing the most recent 10 out of 27 publications