HSV-1 Gene Expression in Latency Infected Neurons - Lawrence Feldman

Abstract

Funding Agency

Agency: National Institute of Health (NIH)
Institute: National Institute of Allergy and Infectious Diseases (NIAID)
Type: Research Project (R01)
Project #: 2R01AI028338-06A2
Application #: 2064379
Study Section: Virology Study Section (VR)

Project Start: 1989-08-01
Project End: 2001-06-30
Budget Start: 1996-07-01
Budget End: 1997-06-30
Support Year: 6
Fiscal Year: 1996
Total Cost
Indirect Cost

Institution

Name: University of California Los Angeles
Department: Microbiology/Immun/Virology
Type: Schools of Medicine
DUNS #: 119132785

City: Los Angeles
State: CA
Country: United States
Zip Code: 90095

Related projects


NIH 2000 R01 AI	HSV-1 Gene Expression in Latency Infected Neurons Feldman, Lawrence T. / University of California Los Angeles	$251,679
NIH 1999 R01 AI	HSV-1 Gene Expression in Latency Infected Neurons Feldman, Lawrence T. / University of California Los Angeles
NIH 1998 R01 AI	HSV-1 Gene Expression in Latency Infected Neurons Feldman, Lawrence T. / University of California Los Angeles
NIH 1997 R01 AI	HSV-1 Gene Expression in Latency Infected Neurons Feldman, Lawrence T. / University of California Los Angeles
NIH 1996 R01 AI	HSV-1 Gene Expression in Latency Infected Neurons Feldman, Lawrence T. / University of California Los Angeles
NIH 1993 R01 AI	HSV-1 Gene Expression in Latently Infected Neurons Feldman, Lawrence T. / University of California Los Angeles
NIH 1992 R01 AI	HSV-1 Gene Expression in Latently Infected Neurons Feldman, Lawrence T. / University of California Los Angeles
NIH 1991 R01 AI	HSV-1 Gene Expression in Latently Infected Neurons Feldman, Lawrence T. / University of California Los Angeles
NIH 1990 R01 AI	HSV-1 Gene Expression in Latently Infected Neurons Feldman, Lawrence T. / University of California Los Angeles
NIH 1989 R01 AI	HSV-1 Gene Expression in Latently Infected Neurons Feldman, Lawrence T. / University of California Los Angeles

Publications

Berthomme, H; Thomas, J; Texier, P et al. (2001) Enhancer and long-term expression functions of herpes simplex virus type 1 latency-associated promoter are both located in the same region. J Virol 75:4386-93

Berthomme, H; Lokensgard, J; Yang, L et al. (2000) Evidence for a bidirectional element located downstream from the herpes simplex virus type 1 latency-associated promoter that increases its activity during latency. J Virol 74:3613-22

Lokensgard, J R; Berthomme, H; Feldman, L T (1997) The latency-associated promoter of herpes simplex virus type 1 requires a region downstream of the transcription start site for long-term expression during latency. J Virol 71:6714-9

Dobson, A T; Margolis, T P; Gomes, W A et al. (1995) In vivo deletion analysis of the herpes simplex virus type 1 latency-associated transcript promoter. J Virol 69:2264-70

Farrell, M J; Margolis, T P; Gomes, W A et al. (1994) Effect of the transcription start region of the herpes simplex virus type 1 latency-associated transcript promoter on expression of productively infected neurons in vivo. J Virol 68:5337-43

Lokensgard, J R; Bloom, D C; Dobson, A T et al. (1994) Long-term promoter activity during herpes simplex virus latency. J Virol 68:7148-58

Farrell, M J; Hill, J M; Margolis, T P et al. (1993) The herpes simplex virus type 1 reactivation function lies outside the latency-associated transcript open reading frame ORF-2. J Virol 67:3653-5

Farrell, M J; Dobson, A T; Feldman, L T (1991) Herpes simplex virus latency-associated transcript is a stable intron. Proc Natl Acad Sci U S A 88:790-4

Dobson, A T; Margolis, T P; Sedarati, F et al. (1990) A latent, nonpathogenic HSV-1-derived vector stably expresses beta-galactosidase in mouse neurons. Neuron 5:353-60

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