Mechanisms of Hematological Toxicity of Antiaids Drugs

Agrawal, Krishna C.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Allergy and Infectious Diseases (NIAID)
• Type: Research Project (R01)
• Project #: 2R01AI032893-04
• Application #: 2067782
• Study Section: Special Emphasis Panel (ZRG5-ARRB (01))
• Project Start: 1993-04-01
• Project End: 1999-03-31
• Budget Start: 1996-04-01
• Budget End: 1997-03-31
• Support Year: 4
• Fiscal Year: 1996

Institution

• Name: Tulane University
• Department: Pharmacology
• Type: Schools of Medicine
• City: New Orleans
• State: LA
• Country: United States
• Zip Code: 70118

Publications

Abdel-Mageed, Asim B; Zhao, Fusheng; Rider, Barbara J et al. (2003) Erythropoietin-induced metallothionein gene expression: role in proliferation of K562 cells. Exp Biol Med (Maywood) 228:1033-9

Chitnis, Shilpa; Mondal, Debasis; Agrawal, Krishna C (2002) Zidovudine (AZT) treatment suppresses granulocyte-monocyte colony stimulating factor receptor type alpha (GM-CSFR alpha) gene expression in murine bone marrow cells. Life Sci 71:967-78

Mondal, D; Agrawal, K C (1996) Effect of HIV type 1 Tat protein on butyric acid-induced differentiation in a hematopoietic progenitor cell line. AIDS Res Hum Retroviruses 12:1529-36

Gogu, S R; Beckman, B S; Wilson, R B et al. (1995) Inhibitory effects of zidovudine in erythroid progenitor cells. Reversal with a combination of erythropoietin and interleukin-3. Biochem Pharmacol 50:413-9