Leishmania Antigen Presentation by Infected Cells

Kima, Peter E.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Allergy and Infectious Diseases (NIAID)
• Type: Research Project (R01)
• Project #: 5R01AI048739-02
• Application #: 6805212
• Study Section: Tropical Medicine and Parasitology Study Section (TMP)
• Program Officer: Wali, Tonu M
• Project Start: 2003-09-30
• Project End: 2008-01-31
• Budget Start: 2004-02-01
• Budget End: 2005-01-31
• Support Year: 2
• Fiscal Year: 2004
• Total Cost: $217,500

Institution

• Name: University of Florida
• Department: Microbiology/Immun/Virology
• Type: Schools of Earth Sciences/Natur
• DUNS #: 969663814
• City: Gainesville
• State: FL
• Country: United States
• Zip Code: 32611

Publications

Kima, Peter E; Bonilla, J Alfredo; Cho, Eumin et al. (2010) Identification of Leishmania proteins preferentially released in infected cells using change mediated antigen technology (CMAT). PLoS Negl Trop Dis 4:

Ruhland, Aaron; Kima, Peter E (2009) Activation of PI3K/Akt signaling has a dominant negative effect on IL-12 production by macrophages infected with Leishmania amazonensis promastigotes. Exp Parasitol 122:28-36

Kima, Peter E (2007) The amastigote forms of Leishmania are experts at exploiting host cell processes to establish infection and persist. Int J Parasitol 37:1087-96

Kima, Peter E; Dunn, Waltraud (2005) Exploiting calnexin expression on phagosomes to isolate Leishmania parasitophorous vacuoles. Microb Pathog 38:139-45

Pham, Nam-Kha; Mouriz, Jennifer; Kima, Peter E (2005) Leishmania pifanoi amastigotes avoid macrophage production of superoxide by inducing heme degradation. Infect Immun 73:8322-33