Cartilage Autocatabolism in Infectious Arthritis

Schurman, David J.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS)
• Type: Research Project (R01)
• Project #: 3R01AR030796-02S1
• Application #: 3155893
• Study Section: Orthopedics and Musculoskeletal Study Section (ORTH)
• Project Start: 1984-06-01
• Project End: 1986-05-31
• Budget Start: 1986-04-01
• Budget End: 1986-05-31
• Support Year: 2
• Fiscal Year: 1986

Institution

• Name: Stanford University
• Type: Schools of Medicine
• DUNS #: 800771545
• City: Stanford
• State: CA
• Country: United States
• Zip Code: 94305

Publications

Mohtai, M; Smith, R L; Schurman, D J et al. (1993) Expression of 92-kD type IV collagenase/gelatinase (gelatinase B) in osteoarthritic cartilage and its induction in normal human articular cartilage by interleukin 1. J Clin Invest 92:179-85

Lane, N E; Williams 3rd, R J; Schurman, D J et al. (1992) Inhibition of interleukin 1 induced chondrocyte protease activity by a corticosteroid and a nonsteroidal antiinflammatory drug. J Rheumatol 19:135-9

Williams 3rd, R J; Smith, R L; Schurman, D J (1991) Purified staphylococcal culture medium stimulates neutral metalloprotease secretion from human articular cartilage. J Orthop Res 9:258-65

Smith, R L (1990) Soluble mediators of articular cartilage degradation in juvenile rheumatoid arthritis. Clin Orthop Relat Res :31-7

Williams 3rd, R J; Smith, R L; Schurman, D J (1990) Septic arthritis. Staphylococcal induction of chondrocyte proteolytic activity. Arthritis Rheum 33:533-41

Smith, R L; Allison, A C; Schurman, D J (1989) Induction of articular cartilage degradation by recombinant interleukin 1 alpha and 1 beta. Connect Tissue Res 18:307-16