Inhibition of Tumor Cell Growth by the Lymphokine Sirs

Schnaper, H William

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Cancer Institute (NCI)
• Type: Research Project (R01)
• Project #: 7R01CA040567-03
• Application #: 3180700
• Study Section: Experimental Immunology Study Section (EI)
• Project Start: 1988-02-01
• Project End: 1990-01-31
• Budget Start: 1988-02-01
• Budget End: 1990-01-31
• Support Year: 3
• Fiscal Year: 1988

Institution

• Name: Washington University
• Type: Schools of Medicine
• DUNS #: 062761671
• City: Saint Louis
• State: MO
• Country: United States
• Zip Code: 63130

Publications

Schnaper, H W (1990) A regulatory system for soluble immune response suppressor production in steroid-responsive nephrotic syndrome. Kidney Int 38:151-9

Schnaper, H W (1989) Divalent metal requirement for soluble immune response suppressor (SIRS) activity. Cell Immunol 118:157-65

Schnaper, H W; Aune, T M; Roby, R K (1987) A role for histamine type II (H-2) receptor binding in production of the lymphokine, soluble immune response suppressor (SIRS). J Immunol 139:1185-90