Modulation of BCL-2 Expression by Class Switch Deletion

Zelenetz, Andrew D.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Cancer Institute (NCI)
• Type: Research Project (R01)
• Project #: 5R01CA058842-04
• Application #: 2099473
• Study Section: Pathology B Study Section (PTHB)
• Project Start: 1993-07-01
• Project End: 1997-04-30
• Budget Start: 1996-08-15
• Budget End: 1997-04-30
• Support Year: 4
• Fiscal Year: 1996

Institution

• Name: Sloan-Kettering Institute for Cancer Research
• DUNS #: 064931884
• City: New York
• State: NY
• Country: United States
• Zip Code: 10065

Publications

Klampfer, L; Zhang, J; Zelenetz, A O et al. (1996) The AML1/ETO fusion protein activates transcription of BCL-2. Proc Natl Acad Sci U S A 93:14059-64

Tsang, P; Gilles, F; Yuan, L et al. (1995) A novel L23-related gene 40 kb downstream of the imprinted H19 gene is biallelically expressed in mid-fetal and adult human tissues. Hum Mol Genet 4:1499-507

Goy, A; Passalaris, T; Xiao, Y H et al. (1995) The PML gene is linked to a megabase-scale insertion/deletion restriction fragment length polymorphism. Genomics 26:327-33