Variation, Immune Escape, and Pathogenesis of Mulv - William Green

Abstract

Funding Agency

Agency: National Institute of Health (NIH)
Institute: National Cancer Institute (NCI)
Type: Research Project (R01)
Project #: 1R01CA069525-01
Application #: 2113584
Study Section: Virology Study Section (VR)

Project Start: 1996-04-01
Project End: 2001-01-31
Budget Start: 1996-04-01
Budget End: 1997-01-31
Support Year: 1
Fiscal Year: 1996
Total Cost
Indirect Cost

Institution

Name: Dartmouth College
Department: Microbiology/Immun/Virology
Type: Schools of Medicine
DUNS #: 041027822

City: Hanover
State: NH
Country: United States
Zip Code: 03755

Related projects


NIH 2000 R01 CA	Variation, Immune Escape, and Pathogenesis of Mulv Green, William R. / Dartmouth College	$233,873
NIH 1999 R01 CA	Variation, Immune Escape, and Pathogenesis of Mulv Green, William R. / Dartmouth College
NIH 1998 R01 CA	Variation, Immune Escape, and Pathogenesis of Mulv Green, William R. / Dartmouth College
NIH 1997 R01 CA	Variation, Immune Escape, and Pathogenesis of Mulv Green, William R. / Dartmouth College
NIH 1996 R01 CA	Variation, Immune Escape, and Pathogenesis of Mulv Green, William R. / Dartmouth College

Publications

Gaur, Arti; Green, William R (2003) Analysis of the helper virus in murine retrovirus-induced immunodeficiency syndrome: evidence for immunoselection of the dominant and subdominant CTL epitopes of the BM5 ecotropic virus. Viral Immunol 16:203-12

Rich, Robert F; Green, William R (2002) Characterization of the Fas ligand/Fas-dependent apoptosis of antiretroviral, class I MHC tetramer-defined, CD8+ CTL by in vivo retrovirus-infected cells. J Immunol 168:2751-8

Kim, V; Yewdell, J W; Green, W R (2000) Naturally occurring TAP-dependent specific T-cell tolerance for a variant of an immunodominant retroviral cytotoxic T-lymphocyte epitope. J Virol 74:3924-8

Green, W R (1999) Cytotoxic T lymphocytes to endogenous mouse retroviruses and mechanisms of retroviral escape. Immunol Rev 168:271-86

Rich, R F; Green, W R (1999) Antiretroviral cytolytic T-lymphocyte nonresponsiveness: FasL/Fas-mediated inhibition of CD4(+) and CD8(+) antiviral T cells by viral antigen-positive veto cells. J Virol 73:3826-34

Kim, V; Green, W R (1998) A single amino acid variation within an immunodominant AKR/Gross MuLV cytotoxic T-lymphocyte epitope leads to a loss in immunogenicity. Viral Immunol 11:197-213

Kim, V; Green, W R (1997) The role of proximal and distal sequence variations in the presentation of an immunodominant CTL epitope encoded by the ecotropic AK7 MuLV. Virology 236:221-33

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