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Eisch, A J; Marshall, J F (1998) Methamphetamine neurotoxicity: dissociation of striatal dopamine terminal damage from parietal cortical cell body injury. Synapse 30:433-45
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Eisch, A J; Schmued, L C; Marshall, J F (1998) Characterizing cortical neuron injury with Fluoro-Jade labeling after a neurotoxic regimen of methamphetamine. Synapse 30:329-33
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Vargo, J M; Marshall, J F (1996) Frontal cortex ablation reversibly decreases striatal zif/268 and junB expression: temporal correspondence with sensory neglect and its spontaneous recovery. Synapse 22:291-303
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LaHoste, G J; Ruskin, D N; Marshall, J F (1996) Cerebrocortical Fos expression following dopaminergic stimulation: D1/D2 synergism and its breakdown. Brain Res 728:97-104
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Vargo, J M; Marshall, J F (1996) Unilateral frontal cortex ablation producing neglect causes time-dependent changes in striatal glutamate receptors. Behav Brain Res 77:189-99
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Vargo, J M; Bromberg, B B; Best, P J et al. (1995) D1-class dopamine receptor involvement in the behavioral recovery from prefrontal cortical injury. Behav Brain Res 72:39-48
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Vargo, J M; Marshall, J F (1995) Time-dependent changes in dopamine agonist-induced striatal Fos immunoreactivity are related to sensory neglect and its recovery after unilateral prefrontal cortex injury. Synapse 20:305-15
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O'Dell, S J; Weihmuller, F B; McPherson, R J et al. (1994) Excitotoxic striatal lesions protect against subsequent methamphetamine-induced dopamine depletions. J Pharmacol Exp Ther 269:1319-25
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Ruskin, D N; Marshall, J F (1994) Amphetamine- and cocaine-induced fos in the rat striatum depends on D2 dopamine receptor activation. Synapse 18:233-40
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Weihmuller, F B; O'Dell, S J; Marshall, J F (1993) L-dopa pretreatment potentiates striatal dopamine overflow and produces dopamine terminal injury after a single methamphetamine injection. Brain Res 623:303-7
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