Agency
National Institute of Health (NIH)
Institute
National Institute on Deafness and Other Communication Disorders (NIDCD)
Type
Research Project (R01)
Project #
5R01DC002120-02
Application #
2127252
Study Section
Sensory Disorders and Language Study Section (CMS)
Project Start
1995-01-01
Project End
1999-12-31
Budget Start
1996-01-01
Budget End
1996-12-31
Support Year
2
Fiscal Year
1996
Total Cost
Indirect Cost
Name
University of Texas Sw Medical Center Dallas
Department
Physiology
Type
Schools of Medicine
DUNS #
City
Dallas
State
TX
Country
United States
Zip Code
75390
Gu, J; Dudley, C; Su, T et al. (1999) Cytochrome P450 and steroid hydroxylase activity in mouse olfactory and vomeronasal mucosa. Biochem Biophys Res Commun 266:262-7
Moss, R L; Flynn, R E; Shi, J et al. (1998) Electrophysiological and biochemical responses of mouse vomeronasal receptor cells to urine-derived compounds: possible mechanism of action. Chem Senses 23:483-9
Moss, R L; Flynn, R E; Shen, X M et al. (1997) Urine-derived compound evokes membrane responses in mouse vomeronasal receptor neurons. J Neurophysiol 77:2856-62
Guo, J; Zhou, A; Moss, R L (1997) Urine and urine-derived compounds induce c-fos mRNA expression in accessory olfactory bulb. Neuroreport 8:1679-83
Zhou, A; Moss, R L (1997) Effect of urine-derived compounds on cAMP accumulation in mouse vomeronasal cells. Neuroreport 8:2173-7
Dudley, C A; Rajendren, G; Moss, R L (1996) Signal processing in the vomeronasal system: modulation of sexual behavior in the female rat. Crit Rev Neurobiol 10:265-90