Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
Research Project (R01)
Project #
5R01DK034028-09
Application #
2139210
Study Section
Experimental Immunology Study Section (EI)
Project Start
1983-12-01
Project End
1998-06-30
Budget Start
1996-07-01
Budget End
1998-06-30
Support Year
9
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Beth Israel Deaconess Medical Center
Department
Type
DUNS #
076593722
City
Boston
State
MA
Country
United States
Zip Code
02215
Klickstein, L B; Barbashov, S F; Liu, T et al. (1997) Complement receptor type 1 (CR1, CD35) is a receptor for C1q. Immunity 7:345-55
Wang, C; Gerard, N P; Nicholson-Weller, A (1996) Signaling by hemolytically inactive C5b67, an agonist of polymorphonuclear leukocytes. J Immunol 156:786-92
Jack, R M; Lowenstein, B A; Nicholson-Weller, A (1994) Regulation of C1q receptor expression on human polymorphonuclear leukocytes. J Immunol 153:262-9
Nicholson-Weller, A; Wang, C E (1994) Structure and function of decay accelerating factor CD55. J Lab Clin Med 123:485-91
Landa, A; Laclette, J P; Nicholson-Weller, A et al. (1993) cDNA cloning and recombinant expression of collagen-binding and complement inhibitor activity of Taenia solium paramyosin (AgB). Mol Biochem Parasitol 60:343-7
Nicholson-Weller, A; Halperin, J A (1993) Membrane signaling by complement C5b-9, the membrane attack complex. Immunol Res 12:244-57
Lopez-Trascasa, M; Bing, D H; Rivard, M et al. (1989) Factor J: isolation and characterization of a new polypeptide inhibitor of complement C1. J Biol Chem 264:16214-21