Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
Research Project (R01)
Project #
5R01DK047990-03
Application #
2147981
Study Section
Pathology B Study Section (PTHB)
Project Start
1994-06-01
Project End
1999-05-31
Budget Start
1996-06-01
Budget End
1997-05-31
Support Year
3
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
University of Arkansas for Medical Sciences
Department
Internal Medicine/Medicine
Type
Schools of Medicine
DUNS #
City
Little Rock
State
AR
Country
United States
Zip Code
72205

  Publications

Basnakian, Alexei G; Ueda, Norishi; Kaushal, Gur P et al. (2002) DNase I-like endonuclease in rat kidney cortex that is activated during ischemia/reperfusion injury. J Am Soc Nephrol 13:1000-7
Carmago, Simone; Shah, Sudhir V; Walker, Patrick D (2002) Meprin, a brush-border enzyme, plays an important role in hypoxic/ischemic acute renal tubular injury in rats. Kidney Int 61:959-66
Ueda, N; Shah, S V (2000) Tubular cell damage in acute renal failure-apoptosis, necrosis, or both. Nephrol Dial Transplant 15:318-23
Ueda, N; Shah, S V (2000) Role of endonucleases in renal tubular epithelial cell injury. Exp Nephrol 8:13-Aug
Ueda, N; Kaushal, G P; Shah, S V (2000) Apoptotic mechanisms in acute renal failure. Am J Med 108:403-15
Ueda, N; Kaushal, G P; Shah, S V (1997) Recent advances in understanding mechanisms of renal tubular injury. Adv Ren Replace Ther 4:17-24
Kaushal, G P; Ueda, N; Shah, S V (1997) Role of caspases (ICE/CED 3 proteases) in DNA damage and cell death in response to a mitochondrial inhibitor, antimycin A. Kidney Int 52:438-45
Hagar, H; Ueda, N; Shah, S V (1996) Role of reactive oxygen metabolites in DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells. Am J Physiol 271:F209-15
Hagar, H; Ueda, N; Shah, S V (1996) Endonuclease induced DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells. Kidney Int 49:355-61