Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
Research Project (R01)
Project #
5R01DK050830-10
Application #
2151905
Study Section
Diabetes, Endocrinology and Metabolic Diseases B Subcommittee (DDK)
Project Start
1987-04-01
Project End
1997-03-31
Budget Start
1996-04-01
Budget End
1997-03-31
Support Year
10
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
University of Alabama Birmingham
Department
Physiology
Type
Schools of Dentistry
DUNS #
004514360
City
Birmingham
State
AL
Country
United States
Zip Code
35294

  Publications

Kirk, K L (2000) New paradigms of CFTR chloride channel regulation. Cell Mol Life Sci 57:623-34
Naren, A P; Di, A; Cormet-Boyaka, E et al. (2000) Syntaxin 1A is expressed in airway epithelial cells, where it modulates CFTR Cl(-) currents. J Clin Invest 105:377-86
Naren, A P; Cormet-Boyaka, E; Fu, J et al. (1999) CFTR chloride channel regulation by an interdomain interaction. Science 286:544-8
Naren, A P; Quick, M W; Collawn, J F et al. (1998) Syntaxin 1A inhibits CFTR chloride channels by means of domain-specific protein-protein interactions. Proc Natl Acad Sci U S A 95:10972-7
Jilling, T; Kirk, K L (1996) Cyclic AMP and chloride-dependent regulation of the apical constitutive secretory pathway in colonic epithelial cells. J Biol Chem 271:4381-7
Weber, E; Jilling, T; Kirk, K L (1996) Distinct functional properties of Rab3A and Rab3B in PC12 neuroendocrine cells. J Biol Chem 271:6963-71