Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Research Project (R01)
Project #
5R01GM029135-10
Application #
3276644
Study Section
Cellular Biology and Physiology Subcommittee 1 (CBY)
Project Start
1981-01-01
Project End
1992-09-14
Budget Start
1992-07-01
Budget End
1992-09-14
Support Year
10
Fiscal Year
1992
Total Cost
Indirect Cost

  Institution

Name
Northwestern University at Chicago
Department
Type
Schools of Dentistry
DUNS #
005436803
City
Chicago
State
IL
Country
United States
Zip Code
60611

  Publications

Leibovich, S J; Polverini, P J; Fong, T W et al. (1994) Production of angiogenic activity by human monocytes requires an L-arginine/nitric oxide-synthase-dependent effector mechanism. Proc Natl Acad Sci U S A 91:4190-4
Koch, A E; Nickoloff, B J; Holgersson, J et al. (1994) 4A11, a monoclonal antibody recognizing a novel antigen expressed on aberrant vascular endothelium. Upregulation in an in vivo model of contact dermatitis. Am J Pathol 144:244-59
Koch, A E; Cho, M; Burrows, J C et al. (1992) Inhibition of production of monocyte/macrophage-derived angiogenic activity by oxygen free-radical scavengers. Cell Biol Int Rep 16:415-25
Koch, A E; Burrows, J C; Haines, G K et al. (1991) Immunolocalization of endothelial and leukocyte adhesion molecules in human rheumatoid and osteoarthritic synovial tissues. Lab Invest 64:313-20
Wiseman, D M; Polverini, P J; Kamp, D W et al. (1988) Transforming growth factor-beta (TGF beta) is chemotactic for human monocytes and induces their expression of angiogenic activity. Biochem Biophys Res Commun 157:793-800
Koch, A E; Polverini, P J; Leibovich, S J (1986) Stimulation of neovascularization by human rheumatoid synovial tissue macrophages. Arthritis Rheum 29:471-9
Koch, A E; Polverini, P J; Leibovich, S J (1986) Induction of neovascularization by activated human monocytes. J Leukoc Biol 39:233-8
Polverini, P J; Leibovich, S J (1985) Induction of neovascularization and nonlymphoid mesenchymal cell proliferation by macrophage cell lines. J Leukoc Biol 37:279-88