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Molecular Basis for Selectivity & Transport in Ion Pumps
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Abstract
Funding Agency
Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Research Project (R01)
Project #
5R01GM052414-03
Application #
2899288
Study Section
Project Start
1998-01-01
Project End
1998-08-31
Budget Start
Budget End
Support Year
3
Fiscal Year
1999
Total Cost
Indirect Cost
Related projects
NIH 1999
R01 GM
Molecular Basis for Selectivity & Transport in Ion Pumps
Rao, Rajini / Johns Hopkins University
NIH 1999
R01 GM
Molecular Basis for Selectivity & Transport in Ion Pumps
/
NIH 1998
R01 GM
Molecular Basis for Selectivity & Transport in Ion Pumps
Rao, Rajini / Johns Hopkins University
NIH 1998
R01 GM
Molecular Basis for Selectivity & Transport in Ion Pumps
Rao, Rajini / Johns Hopkins University
NIH 1997
R01 GM
Molecular Basis for Selectivity & Transport in Ion Pumps
Rao, Rajini / Johns Hopkins University
NIH 1996
R01 GM
Molecular Basis for Selectivity & Transport in Ion Pumps
Rao, Rajini / Johns Hopkins University
Publications
Patenaude, Cassandra; Zhang, Yongqiang; Cormack, Brendan et al.
(2013)
Essential role for vacuolar acidification in Candida albicans virulence.
J Biol Chem 288:26256-64
Leitch, Sharon; Feng, Mingye; Muend, Sabina et al.
(2011)
Vesicular distribution of Secretory Pathway Ca²+-ATPase isoform 1 and a role in manganese detoxification in liver-derived polarized cells.
Biometals 24:159-70
Mandal, D; Woolf, T B; Rao, R
(2000)
Manganese selectivity of pmr1, the yeast secretory pathway ion pump, is defined by residue gln783 in transmembrane segment 6. Residue Asp778 is essential for cation transport.
J Biol Chem 275:23933-8
Wei, Y; Chen, J; Rosas, G et al.
(2000)
Phenotypic screening of mutations in Pmr1, the yeast secretory pathway Ca2+/Mn2+-ATPase, reveals residues critical for ion selectivity and transport.
J Biol Chem 275:23927-32
Marchi, V; Sorin, A; Wei, Y et al.
(1999)
Induction of vacuolar Ca2+-ATPase and H+/Ca2+ exchange activity in yeast mutants lacking Pmr1, the Golgi Ca2+-ATPase.
FEBS Lett 454:181-6
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