Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Research Project (R01)
Project #
5R01GM054351-02
Application #
2193722
Study Section
Allergy and Immunology Study Section (ALY)
Project Start
1995-08-01
Project End
2000-07-31
Budget Start
1996-08-01
Budget End
1997-07-31
Support Year
2
Fiscal Year
1996
Total Cost
Indirect Cost
Name
J. David Gladstone Institutes
Department
Type
DUNS #
047120084
City
San Francisco
State
CA
Country
United States
Zip Code
94158
Mohibullah, Neeman; Hahn, Steven (2008) Site-specific cross-linking of TBP in vivo and in vitro reveals a direct functional interaction with the SAGA subunit Spt3. Genes Dev 22:2994-3006
Osborne, Lisa C; Dhanji, Salim; Snow, Jonathan W et al. (2007) Impaired CD8 T cell memory and CD4 T cell primary responses in IL-7R alpha mutant mice. J Exp Med 204:619-31
Abraham, Ninan; Ma, Melissa C; Snow, Jonathan W et al. (2005) Haploinsufficiency identifies STAT5 as a modifier of IL-7-induced lymphomas. Oncogene 24:5252-7
Snow, Jonathan W; Abraham, Ninan; Ma, Melissa C et al. (2003) Transgenic bcl-2 is not sufficient to rescue all hematolymphoid defects in STAT5A/5B-deficient mice. Exp Hematol 31:1253-8
Snow, Jonathan W; Abraham, Ninan; Ma, Melissa C et al. (2003) Loss of tolerance and autoimmunity affecting multiple organs in STAT5A/5B-deficient mice. J Immunol 171:5042-50
Snow, Jonathan W; Abraham, Ninan; Ma, Melissa C et al. (2003) Bone marrow transplant completely rescues hematolymphoid defects in STAT5A/5B-deficient mice. Exp Hematol 31:1247-52
Usacheva, Anna; Sandoval, Raudel; Domanski, Paul et al. (2002) Contribution of the Box 1 and Box 2 motifs of cytokine receptors to Jak1 association and activation. J Biol Chem 277:48220-6
Snow, Jonathan W; Abraham, Ninan; Ma, Melissa C et al. (2002) STAT5 promotes multilineage hematolymphoid development in vivo through effects on early hematopoietic progenitor cells. Blood 99:95-101
Gaffen, S L; Lai, S Y; Longmore, G D et al. (1999) Genetic evidence for an additional factor required for erythropoietin-induced signal transduction. Blood 94:74-86
Middleton, S A; Barbone, F P; Johnson, D L et al. (1999) Shared and unique determinants of the erythropoietin (EPO) receptor are important for binding EPO and EPO mimetic peptide. J Biol Chem 274:14163-9

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