Monocyte Chemoattractant Protein 1 in the Corpus Luteum

Keyes, Paul Landis

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: Eunice Kennedy Shriver National Institute of Child Health & Human Development (NICHD)
• Type: Research Project (R01)
• Project #: 1R01HD033478-01A1
• Application #: 2206973
• Study Section: Reproductive Biology Study Section (REB)
• Project Start: 1996-05-01
• Project End: 2000-04-30
• Budget Start: 1996-05-01
• Budget End: 1997-04-30
• Support Year: 1
• Fiscal Year: 1996

Institution

• Name: University of Michigan Ann Arbor
• Department: Physiology
• Type: Schools of Medicine
• DUNS #: 791277940
• City: Ann Arbor
• State: MI
• Country: United States
• Zip Code: 48109

Publications

Port, C B; Bowen, J M; Keyes, P L et al. (2000) Effects of a 3beta-hydroxysteroid dehydrogenase inhibitor on monocyte-macrophage infiltration into rat corpus luteum and on apoptosis: relationship to the luteolytic action of prolactin. J Reprod Fertil 119:93-9

Bowen, J M; Telleria, C M; Towns, R et al. (2000) Downregulation of long-form prolactin receptor mRNA during prolactin-induced luteal regression. Eur J Endocrinol 143:285-92

Bowen, J M; Keyes, P L (2000) Repeated exposure to prolactin is required to induce luteal regression in the hypophysectomized rat. Biol Reprod 63:1179-84

Bowen, J M; Towns, R; Warren, J S et al. (1999) Luteal regression in the normally cycling rat: apoptosis, monocyte chemoattractant protein-1, and inflammatory cell involvement. Biol Reprod 60:740-6

Bowen, J M; Keyes, P L (1999) The proestrous prolactin surge is not the sole initiator of regressive changes in corpora lutea of normally cycling rats. Biol Reprod 61:1208-15

Towns, R; Menon, K M; Brabec, R K et al. (1999) Glucocorticoids stimulate the accumulation of lipids in the rat corpus luteum. Biol Reprod 61:416-21