Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Research Project (R01)
Project #
2R01HL048790-05
Application #
2224869
Study Section
Hematology Subcommittee 2 (HEM)
Project Start
1992-08-01
Project End
2000-06-30
Budget Start
1996-08-01
Budget End
1997-06-30
Support Year
5
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
Fred Hutchinson Cancer Research Center
Department
Type
DUNS #
075524595
City
Seattle
State
WA
Country
United States
Zip Code
98109

  Publications

Bigas, A; Martin, D I; Milner, L A (1998) Notch1 and Notch2 inhibit myeloid differentiation in response to different cytokines. Mol Cell Biol 18:2324-33
Sutherland, H G; Martin, D I; Whitelaw, E (1997) A globin enhancer acts by increasing the proportion of erythrocytes expressing a linked transgene. Mol Cell Biol 17:1607-14
Walters, M; Andrews, N C; Magis, W et al. (1996) Erythroid AP-1/NF-E2 elements vary in their response to NF-E2. Exp Hematol 24:445-52
Martin, D I; Whitelaw, E (1996) The vagaries of variegating transgenes. Bioessays 18:919-23
Magis, W; Martin, D I (1995) HMG-I binds to GATA motifs: implications for an HPFH syndrome. Biochem Biophys Res Commun 214:927-33
Fiering, S; Epner, E; Robinson, K et al. (1995) Targeted deletion of 5'HS2 of the murine beta-globin LCR reveals that it is not essential for proper regulation of the beta-globin locus. Genes Dev 9:2203-13
Walters, M; Martin, D I (1992) Functional erythroid promoters created by interaction of the transcription factor GATA-1 with CACCC and AP-1/NFE-2 elements. Proc Natl Acad Sci U S A 89:10444-8