Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Research Project (R01)
Project #
7R01HL051748-03
Application #
2228670
Study Section
Special Emphasis Panel (ZHL1-CSR-S (S2))
Project Start
1993-12-01
Project End
1997-11-30
Budget Start
1996-01-01
Budget End
1996-11-30
Support Year
3
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Medical College of Wisconsin
Department
Physiology
Type
Schools of Medicine
DUNS #
073134603
City
Milwaukee
State
WI
Country
United States
Zip Code
53226
DeFily, D V; Nishikawa, Y; Chilian, W M (1999) Endothelin antagonists block alpha1-adrenergic constriction of coronary arterioles. Am J Physiol 276:H1028-34
Muller, J M; Davis, M J; Kuo, L et al. (1999) Changes in coronary endothelial cell Ca2+ concentration during shear stress- and agonist-induced vasodilation. Am J Physiol 276:H1706-14
Tiefenbacher, C P; DeFily, D V; Chilian, W M (1998) Requisite role of cardiac myocytes in coronary alpha1-adrenergic constriction. Circulation 98:9-12
Weihrauch, D; Tessmer, J; Warltier, D C et al. (1998) Repetitive coronary artery occlusions induce release of growth factors into the myocardial interstitium. Am J Physiol 275:H969-76
Tiefenbacher, C P; Chilian, W M (1998) Heterogeneity of coronary vasomotion. Basic Res Cardiol 93:446-54
Ramos, K S; Sadhu, D N; Meininger, C J et al. (1997) The anti-mitogenic activity of 17beta-estradiol in coronary smooth muscle cells correlates with protein binding to its responsive element. In Vitro Cell Dev Biol Anim 33:738-41
Muller, J M; Chilian, W M; Davis, M J (1997) Integrin signaling transduces shear stress--dependent vasodilation of coronary arterioles. Circ Res 80:320-6