Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Research Project (R01)
Project #
5R01HL053918-03
Application #
2232057
Study Section
Special Emphasis Panel (ZHL1-CSR-S (S2))
Project Start
1994-08-01
Project End
1998-07-31
Budget Start
1996-08-01
Budget End
1997-07-31
Support Year
3
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Northwestern University at Chicago
Department
Pediatrics
Type
Schools of Medicine
DUNS #
005436803
City
Chicago
State
IL
Country
United States
Zip Code
60611
Groten, Tanja; Pierce, Amy A; Huen, Arthur C et al. (2005) 17 beta-estradiol transiently disrupts adherens junctions in endothelial cells. FASEB J 19:1368-70
Chavers, B; Schnaper, H W (2001) Risk factors for cardiovascular disease in children on maintenance dialysis. Adv Ren Replace Ther 8:180-90
Albuquerque, M L; Waters, C M; Savla, U et al. (2000) Shear stress enhances human endothelial cell wound closure in vitro. Am J Physiol Heart Circ Physiol 279:H293-302
Schnaper, H W; McGuire, J; Runyan, C et al. (2000) Sex steroids and the endothelium. Curr Med Chem 7:519-31
Schnaper, H W (1999) Estrogen: it's not just for reproduction any more. Kidney Int 55:1577-9
Poncelet, A C; de Caestecker, M P; Schnaper, H W (1999) The transforming growth factor-beta/SMAD signaling pathway is present and functional in human mesangial cells. Kidney Int 56:1354-65
Kim-Schulze, S; Lowe Jr, W L; Schnaper, H W (1998) Estrogen stimulates delayed mitogen-activated protein kinase activity in human endothelial cells via an autocrine loop that involves basic fibroblast growth factor. Circulation 98:413-21
Albuquerque, M L; Akiyama, S K; Schnaper, H W (1998) Basic fibroblast growth factor release by human coronary artery endothelial cells is enhanced by matrix proteins, 17beta-estradiol, and a PKC signaling pathway. Exp Cell Res 245:163-9
Eddy, A A; Schnaper, H W (1998) The nephrotic syndrome: from the simple to the complex. Semin Nephrol 18:304-16
Schnaper, H W; McGowan, K A; Kim-Schulze, S et al. (1996) Oestrogen and endothelial cell angiogenic activity. Clin Exp Pharmacol Physiol 23:247-50

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