Antihypertensive Effect of H,K Atpase Inhibition

Young, David B.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Heart, Lung, and Blood Institute (NHLBI)
• Type: Research Project (R01)
• Project #: 5R01HL054032-02
• Application #: 2232217
• Study Section: Cardiovascular and Renal Study Section (CVB)
• Project Start: 1995-05-01
• Project End: 1998-04-30
• Budget Start: 1996-05-01
• Budget End: 1997-04-30
• Support Year: 2
• Fiscal Year: 1996

Institution

• Name: University of Mississippi Medical Center
• Department: Physiology
• Type: Schools of Medicine
• DUNS #: 928824473
• City: Jackson
• State: MS
• Country: United States
• Zip Code: 39216

Publications

Ma, G; Young, D B; Clower, B R et al. (2000) High potassium intake inhibits neointima formation in the rat carotid artery balloon injury model. Am J Hypertens 13:1014-20

Ma, G; Mason, D P; Young, D B (2000) Inhibition of vascular smooth muscle cell migration by elevation of extracellular potassium concentration. Hypertension 35:948-51

Ma, G; Mamaril, J L; Young, D B (2000) Increased potassium concentration inhibits stimulation of vascular smooth muscle proliferation by PDGF-BB and bFGF. Am J Hypertens 13:1055-60

Young, D B; Ma, G (1999) Vascular protective effects of potassium. Semin Nephrol 19:477-86

Lin, H; Young, D B (1997) Reduction in renin release and renal vascular resistance by H(+)-K(+)-ATPase inhibition. Am J Physiol 273:F457-62