Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
First Independent Research Support & Transition (FIRST) Awards (R29)
Project #
5R29HL047432-04
Application #
2223653
Study Section
Experimental Cardiovascular Sciences Study Section (ECS)
Project Start
1993-09-01
Project End
1998-08-31
Budget Start
1996-09-01
Budget End
1997-08-31
Support Year
4
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Northeast Ohio Medical University
Department
Physiology
Type
Schools of Medicine
DUNS #
City
Rootstown
State
OH
Country
United States
Zip Code
44272
Li, Min; Kuo, Lih; Stallone, John N (2008) Estrogen potentiates constrictor prostanoid function in female rat aorta by upregulation of cyclooxygenase-2 and thromboxane pathway expression. Am J Physiol Heart Circ Physiol 294:H2444-55
Sellers, Minga M; Stallone, John N (2008) Sympathy for the devil: the role of thromboxane in the regulation of vascular tone and blood pressure. Am J Physiol Heart Circ Physiol 294:H1978-86
Li, Min; Stallone, John N (2005) Estrogen potentiates vasopressin-induced contraction of female rat aorta by enhancing cyclooxygenase-2 and thromboxane function. Am J Physiol Heart Circ Physiol 289:H1542-50
Voss, M R; Stallone, J N; Li, Min et al. (2003) Gender differences in the expression of heat shock proteins: the effect of estrogen. Am J Physiol Heart Circ Physiol 285:H687-92
Fulton, Clifford T; Stallone, John N (2002) Sexual dimorphism in prostanoid-potentiated vascular contraction: roles of endothelium and ovarian steroids. Am J Physiol Heart Circ Physiol 283:H2062-73
Stallone, J N; Salisbury, R L; Fulton, C T (2001) Androgen-receptor defect abolishes sex differences in nitric oxide and reactivity to vasopressin in rat aorta. J Appl Physiol 91:2602-10
Deenadayalu, V P; White, R E; Stallone, J N et al. (2001) Testosterone relaxes coronary arteries by opening the large-conductance, calcium-activated potassium channel. Am J Physiol Heart Circ Physiol 281:H1720-7
Ding, A Q; Stallone, J N (2001) Testosterone-induced relaxation of rat aorta is androgen structure specific and involves K+ channel activation. J Appl Physiol 91:2742-50
Chao, J; Stallone, J N; Liang, Y M et al. (1997) Kallistatin is a potent new vasodilator. J Clin Invest 100:11-7
Costarella, C E; Stallone, J N; Rutecki, G W et al. (1996) Testosterone causes direct relaxation of rat thoracic aorta. J Pharmacol Exp Ther 277:34-9

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