Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
First Independent Research Support & Transition (FIRST) Awards (R29)
Project #
5R29HL052221-03
Application #
2229484
Study Section
Cardiovascular and Renal Study Section (CVB)
Project Start
1994-04-01
Project End
1999-03-31
Budget Start
1996-04-01
Budget End
1997-03-31
Support Year
3
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Texas Engineering Experiment Station
Department
Physiology
Type
Schools of Engineering
DUNS #
847205572
City
College Station
State
TX
Country
United States
Zip Code
77845
Yuan, S Y (2000) Signal transduction pathways in enhanced microvascular permeability. Microcirculation 7:395-403
Tinsley, J H; Wu, M H; Ma, W et al. (1999) Activated neutrophils induce hyperpermeability and phosphorylation of adherens junction proteins in coronary venular endothelial cells. J Biol Chem 274:24930-4
Wu, H M; Yuan, Y; Zawieja, D C et al. (1999) Role of phospholipase C, protein kinase C, and calcium in VEGF-induced venular hyperpermeability. Am J Physiol 276:H535-42
Yuan, Y; Meng, F Y; Huang, Q et al. (1998) Tyrosine phosphorylation of paxillin/pp125FAK and microvascular endothelial barrier function. Am J Physiol 275:H84-93
Huang, Q; Wu, M; Meininger, C et al. (1998) Neutrophil-dependent augmentation of PAF-induced vasoconstriction and albumin flux in coronary arterioles. Am J Physiol 275:H1138-47
Meng, F; Korompai, F L; Lynch, D M et al. (1998) Acetylcholine-induced and nitric oxide-mediated vasodilation in burns. J Surg Res 80:236-42
Tinsley, J H; Hawker, J; Yuan, Y (1998) Efficient protein transfection of cultured coronary venular endothelial cells. Am J Physiol 275:H1873-8
Yuan, Y; Huang, Q; Wu, H M (1997) Myosin light chain phosphorylation: modulation of basal and agonist-stimulated venular permeability. Am J Physiol 272:H1437-43
Huang, Q; Yuan, Y (1997) Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability. Am J Physiol 273:H2442-51
Wu, H M; Yuan, Y; McCarthy, M et al. (1996) Acidic and basic FGFs dilate arterioles of skeletal muscle through a NO-dependent mechanism. Am J Physiol 271:H1087-93

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