Conceived for highly qualified, exceptional students interested in careers in medical research and academic medicine, the M.D./Ph.D Combined Degree is an intensive seven to eight year program that allows students to acquire clinical skills and competence in the scientific method that can be applied to fundamental problems associated with human disease. The training program meets all of the requirements of the Graduate School for the Doctor of Philosophy Degree, as well as of the School of Medicine for the Doctor of Medicine Degree. The intent of the Combined Degree Program is to allow a student to combine the curricula of the graduate and medical Schools to complete the requirements of both schools in a period less than that required if the graduate and medical studies were taken in sequence. The first two years of the program are devoted to completing the basic medical science curriculum, which includes Mechanisms of Disease, Correlated Medical Problem Solving, Student Continuity Practice, Principles of Clinical Medicine, as well as four Graduate Seminar Courses concerned with Critical Analysis of the Biological Literature and Step I of the United States Medical Licensing Examination. During the next three to four years, the students pursue additional graduate studies and PhD dissertation in one of seven areas of concentration - Cell Biology, Developmental Biology, Immunology, Genetics, Molecular Biology & Biochemistry, Neuroscience, Cellular & Molecular Pharmacology and Biomedical Engineering. Just prior to the thesis defense, students enroll in a five-week Clinical Skills Assessment Program designed to reawaken their history and physical examination skills. Following a successful thesis defense, the students return to the medical school to complete their clinical training. Currently, there are twenty-two students in the Combined Degree Program. These students are selected from a highly competitive applicant pool. With the exception of the Biomedical Engineering Program, in which the University at Storrs participates, the training program occurs entirely at the University of Connecticut Health Center and its affiliated hospitals. ? ?

Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Institutional National Research Service Award (T32)
Project #
5T32GM008607-07
Application #
6915000
Study Section
National Institute of General Medical Sciences Initial Review Group (BRT)
Program Officer
Shapiro, Bert I
Project Start
1999-07-01
Project End
2007-06-30
Budget Start
2005-07-01
Budget End
2006-06-30
Support Year
7
Fiscal Year
2005
Total Cost
$133,435
Indirect Cost
Name
University of Connecticut
Department
Pharmacology
Type
Schools of Medicine
DUNS #
022254226
City
Farmington
State
CT
Country
United States
Zip Code
06030
Kiraly, Drew D; Lemtiri-Chlieh, Fouad; Levine, Eric S et al. (2011) Kalirin binds the NR2B subunit of the NMDA receptor, altering its synaptic localization and function. J Neurosci 31:12554-65
Chan, Grace; Gelernter, Joel; Oslin, David et al. (2011) Empirically derived subtypes of opioid use and related behaviors. Addiction 106:1146-54
Douglas, Kara R; Chan, Grace; Gelernter, Joel et al. (2010) Adverse childhood events as risk factors for substance dependence: partial mediation by mood and anxiety disorders. Addict Behav 35:7-13
Vatner, Ralph E; Srivastava, Pramod K (2010) The tailless complex polypeptide-1 ring complex of the heat shock protein 60 family facilitates cross-priming of CD8 responses specific for chaperoned peptides. J Immunol 185:6765-73
Lauter, Kelly; Arnold, Andrew (2009) Analysis of CYP27B1, encoding 25-hydroxyvitamin D-1alpha-hydroxylase, as a candidate tumor suppressor gene in primary and severe secondary/tertiary hyperparathyroidism. J Bone Miner Res 24:102-4
Lauter, K B; Arnold, A (2008) Mutational analysis of CDKN1B, a candidate tumor-suppressor gene, in refractory secondary/tertiary hyperparathyroidism. Kidney Int 73:1137-40
Flynn, Christopher; Montrose, David C; Swank, Daniel L et al. (2007) Deoxycholic acid promotes the growth of colonic aberrant crypt foci. Mol Carcinog 46:60-70
Niciu, Mark J; Ma, Xin-Ming; El Meskini, Rajaa et al. (2007) Altered ATP7A expression and other compensatory responses in a murine model of Menkes disease. Neurobiol Dis 27:278-91
Costa-Guda, Jessica; Lauter, Kelly; Naveh-Many, Tally et al. (2006) Mutational analysis of the PTH 3'-untranslated region in parathyroid disorders. Clin Endocrinol (Oxf) 65:806-9
Krebs, Linda J; Shattuck, Trisha M; Arnold, Andrew (2005) HRPT2 mutational analysis of typical sporadic parathyroid adenomas. J Clin Endocrinol Metab 90:5015-7

Showing the most recent 10 out of 11 publications