This core unit is located within the administrative offices at the Blood Research Institute, the research arm of the Blood Center of Wisconsin. The BRI includes the offices and laboratories of the research scientists of the institution, but also includes pediatric hematology faculty of the Department of Pediatrics at MCW, and is the location of the Comprehensive Center for Bleeding Disorders. This administrative core will coordinate this multifaceted program and consists of two major components.

Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Research Program Projects (P01)
Project #
7P01HL081588-08
Application #
8608581
Study Section
Heart, Lung, and Blood Initial Review Group (HLBP)
Project Start
Project End
Budget Start
2014-02-01
Budget End
2015-01-31
Support Year
8
Fiscal Year
2014
Total Cost
$1,133,222
Indirect Cost
$53,464
Name
Bloodcenter of Wisconsin, Inc.
Department
Type
DUNS #
057163172
City
Milwaukee
State
WI
Country
United States
Zip Code
53233
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Fahs, Scot A; Hille, Matthew T; Shi, Qizhen et al. (2014) A conditional knockout mouse model reveals endothelial cells as the principal and possibly exclusive source of plasma factor VIII. Blood 123:3706-13
de Brasi, C; El-Maarri, O; Perry, D J et al. (2014) Genetic testing in bleeding disorders. Haemophilia 20 Suppl 4:54-8
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Castaman, G; Hillarp, A; Goodeve, A (2014) Laboratory aspects of von Willebrand disease: test repertoire and options for activity assays and genetic analysis. Haemophilia 20 Suppl 4:65-70
Shida, Yasuaki; Rydz, Natalia; Stegner, David et al. (2014) Analysis of the role of von Willebrand factor, platelet glycoprotein VI-, and ?2?1-mediated collagen binding in thrombus formation. Blood 124:1799-807
Rusu, Luiza; Andreeva, Alexandra; Visintine, David J et al. (2014) G protein-dependent basal and evoked endothelial cell vWF secretion. Blood 123:442-50
Brott, David A; Katein, Anne; Thomas, Heath et al. (2014) Evaluation of von Willebrand factor and von Willebrand factor propeptide in models of vascular endothelial cell activation, perturbation, and/or injury. Toxicol Pathol 42:672-83

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