Administration Core A. Mrs. Kimberly Hagler, Program Administrator, will work with Dr. Timothy Cope, Core A Principal Investigator, and Dr. Robert Fyffe, Core A Co-PI, to establish and manage Program Project business. This office will oversee the day-to-day operations of the Program, including: (1) disseminating all information;(2) organizing and managing group meetings and events;(3) ordering, receiving and distributing supplies;(4) facilitating access to secretarial and computer networking services;(5) assisting personnel actions;(6) maintaining all project accounts and preparing monthly budget reports with the assistance of a part-time accounting clerk;(7) preparing necessary reports for the Program. This core will also manage the subcontract for Project 2 with Emory University, and make all arrangements for the creation and utilization of an External Advisory Committee (EAC) that will meet at Wright State University annually. Decision Making Process is straightforward for this administrative core. Services listed above apply to all 3 Projects and to Core B. Should any special needs or adjustments be required, then all 4 Principal Investigators (projects 1, 2, 3, and Core B) will make the changes by consensus. Cost Effectiveness and Quality Control: The operating costs are heavily shared by the academic infrastructure established for Dept. Neuroscience, Cell Biology &Physiology at Wright State University. Departmental funds cover most of the salary for Mrs. Hagler, together with office space and supplies, computer support, FAX and photocopying. Quality control amounts to oversight by the Core PI, in consultation with the other PIs, of the effectiveness in delivery of services listed above.
This core assumes responsibility for promoting and overseeing activities that are essential to the purpose of the program project mechanism, namely the sharing of ideas, data, and specialized core resources, laboratories, and administrative services.
|Obeidat, Ahmed Z; Nardelli, Paul; Powers, Randall K et al. (2014) Modulation of motoneuron firing by recurrent inhibition in the adult rat in vivo. J Neurophysiol 112:2302-15|
|Rotterman, Travis M; Nardelli, Paul; Cope, Timothy C et al. (2014) Normal distribution of VGLUT1 synapses on spinal motoneuron dendrites and their reorganization after nerve injury. J Neurosci 34:3475-92|
|Deardorff, Adam S; Romer, Shannon H; Sonner, Patrick M et al. (2014) Swimming against the tide: investigations of the C-bouton synapse. Front Neural Circuits 8:106|
|Romer, Shannon H; Dominguez, Kathleen M; Gelpi, Marc W et al. (2014) Redistribution of Kv2.1 ion channels on spinal motoneurons following peripheral nerve injury. Brain Res 1547:1-15|
|Zhang, Jingming; Lanuza, Guillermo M; Britz, Olivier et al. (2014) V1 and v2b interneurons secure the alternating flexor-extensor motor activity mice require for limbed locomotion. Neuron 82:138-50|
|Koesters, Andrew; Engisch, Kathrin L; Rich, Mark M (2014) Decreased cardiac excitability secondary to reduction of sodium current may be a significant contributor to reduced contractility in a rat model of sepsis. Crit Care 18:R54|
|Deardorff, Adam S; Romer, Shannon H; Deng, Zhihui et al. (2013) Expression of postsynaptic Ca2+-activated K+ (SK) channels at C-bouton synapses in mammalian lumbar -motoneurons. J Physiol 591:875-97|
|Nardelli, Paul; Khan, Jaffar; Powers, Randall et al. (2013) Reduced motoneuron excitability in a rat model of sepsis. J Neurophysiol 109:1775-81|
|Wang, Xueyong; Wang, Qingbo; Yang, Shuzhang et al. (2011) Impaired activity-dependent plasticity of quantal amplitude at the neuromuscular junction of Rab3A deletion and Rab3A earlybird mutant mice. J Neurosci 31:3580-8|
|Bullinger, Katie L; Nardelli, Paul; Pinter, Martin J et al. (2011) Permanent central synaptic disconnection of proprioceptors after nerve injury and regeneration. II. Loss of functional connectivity with motoneurons. J Neurophysiol 106:2471-85|
Showing the most recent 10 out of 20 publications