The overall goal of the Education and Information Transfer Core (EITC) is to enhance minority research participation in studies of Mild Cognitive Impairment (MCI), Alzheimer's Disease (AD), and other common dementias by providing multi-culturally sensitive educational services tailored to meet the needs of communities with large numbers of racial and ethnic minorities. The ethnic diversity of metropolitan Chicago including, but not limited to, the presence of large African American communities has shaped the approach to this general goal for the Rush Alzheimer's Disease Center Core (Rush ADCC). The EITC has the responsibility for providing liasion services between the communities served by the Rush ADCC and ADCC researchers. EITC activities will continue to include: 1) networking with community leaders to nurture mutually beneficial long-term relationships;2) """"""""giving first"""""""" to meet the needs of minorities and the communities that serve them;3) advocating for minority research participation;4) """"""""giving back"""""""" through education and outreach by sharing research findings with participants, communities and the professionals serving those communities, and 5) evaluating these activities in order to ensure they address all stakeholder needs and to ensure minority participation in ADCC research projects and National Institute on Aging (NIA) special research initiatives.
By nurturing long-term relationships in multiculturally diverse communities based on mutual trust and respect, the Rush Education and Information Transfer Core impacts minority participation rates in studies of Mild Cognitive Impairment, Alzheimer's disease, and other common dementias. The Rush Education and Information Transfer Core also supports translation of research findings to persons in minority communities.
|Forrester, Sarah N; Gallo, Joseph J; Smith, Gwenn S et al. (2016) Patterns of Neuropsychiatric Symptoms in Mild Cognitive Impairment and Risk of Dementia. Am J Geriatr Psychiatry 24:117-25|
|Burke, Shanna L; Maramaldi, Peter; Cadet, Tamara et al. (2016) Neuropsychiatric symptoms and Apolipoprotein E: Associations with eventual Alzheimer's disease development. Arch Gerontol Geriatr 65:231-8|
|Ibrahim-Verbaas, C A; Bressler, J; Debette, S et al. (2016) GWAS for executive function and processing speed suggests involvement of the CADM2 gene. Mol Psychiatry 21:189-97|
|Wilson, Robert S; Rajan, Kumar B; Barnes, Lisa L et al. (2016) Factors related to racial differences in late-life level of cognitive function. Neuropsychology 30:517-24|
|Mufson, Elliott J; Malek-Ahmadi, Michael; Snyder, Noelle et al. (2016) Braak stage and trajectory of cognitive decline in noncognitively impaired elders. Neurobiol Aging 43:101-10|
|John, Samantha E; Gurnani, Ashita S; Bussell, Cara et al. (2016) The effectiveness and unique contribution of neuropsychological tests and the Î´ latent phenotype in the differential diagnosis of dementia in the uniform data set. Neuropsychology 30:946-960|
|Lim, Andrew S P; Bennett, David A; Buchman, Aron S (2016) Response to Letter Regarding Article, ""Sleep Fragmentation, Cerebral Arteriolosclerosis, and Brain Infarct Pathology in Community-Dwelling Older People"". Stroke 47:e175|
|Montine, Thomas J; Monsell, Sarah E; Beach, Thomas G et al. (2016) Multisite assessment of NIA-AA guidelines for the neuropathologic evaluation of Alzheimer's disease. Alzheimers Dement 12:164-9|
|Dodge, Hiroko H; Zhu, Jian; Woltjer, Randy et al. (2016) Risk of incident clinical diagnosis of Alzheimer's disease-type dementiaÂ attributable to pathology-confirmed vascular disease. Alzheimers Dement :|
|Liu, Rui-Xuan; Huang, Cui; Bennett, David A et al. (2016) The characteristics of astrocyte on AÎ² clearance altered in Alzheimer's disease were reversed by anti-inflammatory agent (+)-2-(1-hydroxyl-4-oxocyclohexyl) ethyl caffeate. Am J Transl Res 8:4082-4094|
Showing the most recent 10 out of 586 publications