The overall goal of the Education and Information Transfer Core (EITC) is to enhance minority research participation in studies of Mild Cognitive Impairment (MCI), Alzheimer's Disease (AD), and other common dementias by providing multi-culturally sensitive educational services tailored to meet the needs of communities with large numbers of racial and ethnic minorities. The ethnic diversity of metropolitan Chicago including, but not limited to, the presence of large African American communities has shaped the approach to this general goal for the Rush Alzheimer's Disease Center Core (Rush ADCC). The EITC has the responsibility for providing liasion services between the communities served by the Rush ADCC and ADCC researchers. EITC activities will continue to include: 1) networking with community leaders to nurture mutually beneficial long-term relationships;2) """"""""giving first"""""""" to meet the needs of minorities and the communities that serve them;3) advocating for minority research participation;4) """"""""giving back"""""""" through education and outreach by sharing research findings with participants, communities and the professionals serving those communities, and 5) evaluating these activities in order to ensure they address all stakeholder needs and to ensure minority participation in ADCC research projects and National Institute on Aging (NIA) special research initiatives.
By nurturing long-term relationships in multiculturally diverse communities based on mutual trust and respect, the Rush Education and Information Transfer Core impacts minority participation rates in studies of Mild Cognitive Impairment, Alzheimer's disease, and other common dementias. The Rush Education and Information Transfer Core also supports translation of research findings to persons in minority communities.
|De Jager, Philip L; Ma, Yiyi; McCabe, Cristin et al. (2018) A multi-omic atlas of the human frontal cortex for aging and Alzheimer's disease research. Sci Data 5:180142|
|Montagne, Axel; Nikolakopoulou, Angeliki M; Zhao, Zhen et al. (2018) Pericyte degeneration causes white matter dysfunction in the mouse central nervous system. Nat Med 24:326-337|
|Barnes, Josephine; Bartlett, Jonathan W; Wolk, David A et al. (2018) Disease Course Varies According to Age and Symptom Length in Alzheimer's Disease. J Alzheimers Dis 64:631-642|
|Dobbyn, Amanda; Huckins, Laura M; Boocock, James et al. (2018) Landscape of Conditional eQTL in Dorsolateral Prefrontal Cortex and Co-localization with Schizophrenia GWAS. Am J Hum Genet 102:1169-1184|
|Wang, Xulong; Philip, Vivek M; Ananda, Guruprasad et al. (2018) A Bayesian Framework for Generalized Linear Mixed Modeling Identifies New Candidate Loci for Late-Onset Alzheimer's Disease. Genetics 209:51-64|
|Alosco, Michael L; Sugarman, Michael A; Besser, Lilah M et al. (2018) A Clinicopathological Investigation of White Matter Hyperintensities and Alzheimer's Disease Neuropathology. J Alzheimers Dis 63:1347-1360|
|Burke, Shanna L; Maramaldi, Peter; Cadet, Tamara et al. (2018) Decreasing hazards of Alzheimer's disease with the use of antidepressants: mitigating the risk of depression and apolipoprotein E. Int J Geriatr Psychiatry 33:200-211|
|Brent, Robert J (2018) Estimating the monetary benefits of medicare eligibility for reducing the symptoms of dementia. Appl Econ 50:6327-6340|
|Chibnik, L B; White, C C; Mukherjee, S et al. (2018) Susceptibility to neurofibrillary tangles: role of the PTPRD locus and limited pleiotropy with other neuropathologies. Mol Psychiatry 23:1521-1529|
|Deming, Yuetiva; Dumitrescu, Logan; Barnes, Lisa L et al. (2018) Sex-specific genetic predictors of Alzheimer's disease biomarkers. Acta Neuropathol 136:857-872|
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