The overall goal of the Education and Information Transfer Core (EITC) is to enhance minority research participation in studies of Mild Cognitive Impairment (MCI), Alzheimer's Disease (AD), and other common dementias by providing multi-culturally sensitive educational services tailored to meet the needs of communities with large numbers of racial and ethnic minorities. The ethnic diversity of metropolitan Chicago including, but not limited to, the presence of large African American communities has shaped the approach to this general goal for the Rush Alzheimer's Disease Center Core (Rush ADCC). The EITC has the responsibility for providing liasion services between the communities served by the Rush ADCC and ADCC researchers. EITC activities will continue to include: 1) networking with community leaders to nurture mutually beneficial long-term relationships;2) "giving first" to meet the needs of minorities and the communities that serve them;3) advocating for minority research participation;4) "giving back" through education and outreach by sharing research findings with participants, communities and the professionals serving those communities, and 5) evaluating these activities in order to ensure they address all stakeholder needs and to ensure minority participation in ADCC research projects and National Institute on Aging (NIA) special research initiatives.
By nurturing long-term relationships in multiculturally diverse communities based on mutual trust and respect, the Rush Education and Information Transfer Core impacts minority participation rates in studies of Mild Cognitive Impairment, Alzheimer's disease, and other common dementias. The Rush Education and Information Transfer Core also supports translation of research findings to persons in minority communities.
|Perez, Sylvia E; He, Bin; Nadeem, Muhammad et al. (2015) Resilience of precuneus neurotrophic signaling pathways despite amyloid pathology in prodromal Alzheimer's disease. Biol Psychiatry 77:693-703|
|Bennett, David A; Yu, Lei; Yang, Jingyun et al. (2015) Epigenomics of Alzheimer's disease. Transl Res 165:200-20|
|Pivtoraiko, Violetta N; Abrahamson, Eric E; Leurgans, Sue E et al. (2015) Cortical pyroglutamate amyloid-? levels and cognitive decline in Alzheimer's disease. Neurobiol Aging 36:9-Dec|
|Ohta, Yasuyuki; Tremblay, Cyntia; Schneider, Julie A et al. (2014) Interaction of transactive response DNA binding protein 43 with nuclear factor ?B in mild cognitive impairment with episodic memory deficits. Acta Neuropathol Commun 2:37|
|Crystal, Howard A; Schneider, Julie A; Bennett, David A et al. (2014) Associations of cerebrovascular and Alzheimer's disease pathology with brain atrophy. Curr Alzheimer Res 11:309-16|
|Verghese, Joe; Annweiler, Cedric; Ayers, Emmeline et al. (2014) Motoric cognitive risk syndrome: multicountry prevalence and dementia risk. Neurology 83:718-26|
|Lurain, Nell S; Hanson, Barbara A; Martinson, Jeffrey et al. (2014) Reply to Itzhaki and Klapper. J Infect Dis 209:974|
|Tangney, Christy C; Li, Hong; Wang, Yamin et al. (2014) Relation of DASH- and Mediterranean-like dietary patterns to cognitive decline in older persons. Neurology 83:1410-6|
|Beach, Thomas G; Schneider, Julie A; Sue, Lucia I et al. (2014) Theoretical impact of Florbetapir (18F) amyloid imaging on diagnosis of alzheimer dementia and detection of preclinical cortical amyloid. J Neuropathol Exp Neurol 73:948-53|
|Nelson, Peter T; Estus, Steven; Abner, Erin L et al. (2014) ABCC9 gene polymorphism is associated with hippocampal sclerosis of aging pathology. Acta Neuropathol 127:825-43|
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