Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Project (R01)
Project #
1R01AI039612-01
Application #
2076661
Study Section
Cellular Biology and Physiology Subcommittee 1 (CBY)
Project Start
1996-06-01
Project End
2001-05-31
Budget Start
1996-06-01
Budget End
1997-05-31
Support Year
1
Fiscal Year
1996
Total Cost
Indirect Cost
Name
University of California Los Angeles
Department
Internal Medicine/Medicine
Type
Schools of Medicine
DUNS #
119132785
City
Los Angeles
State
CA
Country
United States
Zip Code
90095
Gross, Mitchell; Yang, Randy; Top, Irina et al. (2004) PIASy-mediated repression of the androgen receptor is independent of sumoylation. Oncogene 23:3059-66
Liu, B; Shuai, K (2001) Induction of apoptosis by protein inhibitor of activated Stat1 through c-Jun NH2-terminal kinase activation. J Biol Chem 276:36624-31
Liu, B; Gross, M; ten Hoeve, J et al. (2001) A transcriptional corepressor of Stat1 with an essential LXXLL signature motif. Proc Natl Acad Sci U S A 98:3203-7
Graeber, T G; Shuai, K (2000) Rapid gene repression triggered by interleukin-6 at the onset of monocyte differentiation. Biochem Biophys Res Commun 267:863-9
Liao, J; Fu, Y; Shuai, K (2000) Distinct roles of the NH2- and COOH-terminal domains of the protein inhibitor of activated signal transducer and activator of transcription (STAT) 1 (PIAS1) in cytokine-induced PIAS1-Stat1 interaction. Proc Natl Acad Sci U S A 97:5267-72
Liu, B; Liao, J; Rao, X et al. (1998) Inhibition of Stat1-mediated gene activation by PIAS1. Proc Natl Acad Sci U S A 95:10626-31
Chung, C D; Liao, J; Liu, B et al. (1997) Specific inhibition of Stat3 signal transduction by PIAS3. Science 278:1803-5