The Immunology Training Program is the primary training program for Immunology at the University of Chicago and this Immunology Training Grant, currently in its 30th year of funding, represents the primary Training Grant for its graduate students. The Program is conducted in a diverse environment where the basic biological sciences and the medical school and clinical programs are integrated within the same Division in a single Campus in Hyde Park, Chicago, IL. The Program is embodied by the Committee on Immunology, an interdisciplinary and interdepartmental academic unit which includes some of the most distinguished and productive faculty in various Departments. The diverse, gender-balanced faculty is composed of 31 trainers who are highly involved in the Program and whose research spans a broad spectrum of basic and translational Immunology, including emerging areas such as live imaging, genomics and systems biology. Predoctoral students receive advanced training in Immunology along with a core basic curriculum and with electives from a wide range of scientific disciplines. Postdocs receive career development and research training along with training in responsible conduct of research and may take advanced Immunology courses. The comprehensive training also includes a weekly Journal Club, Work in Progress and Seminar Series, an annual Immunology Symposium and a two-day Retreat. The strong institutional support is evidenced by the major allocation of new space and the ongoing regroupment of faculty in a new "Immunology Hub" at the center of the renovated campus. With a world renowned group of highly collaborative faculty, a very large pool of outstanding applicants, a highly successful record of training productive career immunologists including an increasing proportion of underrepresented minorities, this program continues to rank among the best in the country. Training support is requested for ten slots as in the past funding period, with eight predocs and two postdocs for a period of five years.

Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Institutional National Research Service Award (T32)
Project #
5T32AI007090-34
Application #
8277911
Study Section
Special Emphasis Panel (ZAI1-GSM-I (M1))
Program Officer
Prograis, Lawrence J
Project Start
1979-07-01
Project End
2014-06-30
Budget Start
2012-07-01
Budget End
2013-06-30
Support Year
34
Fiscal Year
2012
Total Cost
$475,312
Indirect Cost
$26,213
Name
University of Chicago
Department
Pathology
Type
Schools of Medicine
DUNS #
005421136
City
Chicago
State
IL
Country
United States
Zip Code
60637
Cao, Severine; Feehley, Taylor J; Nagler, Cathryn R (2014) The role of commensal bacteria in the regulation of sensitization to food allergens. FEBS Lett 588:4258-66
Binder, David C; Schreiber, Hans (2014) Dual blockade of PD-1 and CTLA-4 combined with tumor vaccine effectively restores T-cell rejection function in tumors--letter. Cancer Res 74:632; discussion 635
Keerthivasan, Shilpa; Aghajani, Katayoun; Dose, Marei et al. (2014) ?-Catenin promotes colitis and colon cancer through imprinting of proinflammatory properties in T cells. Sci Transl Med 6:225ra28
Zhai, Zili; Wu, Feng; Dong, Fengshi et al. (2014) Human autophagy gene ATG16L1 is post-transcriptionally regulated by MIR142-3p. Autophagy 10:468-79
Wilks, Jessica; Beilinson, Helen; Theriault, Betty et al. (2014) Antibody-mediated immune control of a retrovirus does not require the microbiota. J Virol 88:6524-7
Verykokakis, Mihalis; Zook, Erin C; Kee, Barbara L (2014) ID'ing innate and innate-like lymphoid cells. Immunol Rev 261:177-97
Dose, Marei; Emmanuel, Akinola Olumide; Chaumeil, Julie et al. (2014) *-Catenin induces T-cell transformation by promoting genomic instability. Proc Natl Acad Sci U S A 111:391-6
Stefka, Andrew T; Feehley, Taylor; Tripathi, Prabhanshu et al. (2014) Commensal bacteria protect against food allergen sensitization. Proc Natl Acad Sci U S A 111:13145-50
Mortenson, Eric D; Fu, Yang-Xin (2014) Anti-HER2/Neu passive-aggressive immunotherapy. Oncoimmunology 3:e27296
Chen, Bohao; Moore, Tamson V; Li, Zhenping et al. (2014) Gata5 deficiency causes airway constrictor hyperresponsiveness in mice. Am J Respir Cell Mol Biol 50:787-95

Showing the most recent 10 out of 45 publications