The goal of the Wisconsin Allergy Research Training (WISCART) program is to provide research training for postdoctoral fellows in Allergy and Immunology to prepare trainees for careers as independent academic research scientists. Objectives to achieve this goal include 1) to establish a high quality and productive research project, 2) to develop a progressive record of publication in respected research journals, 3) to obtain extramural grant funding during the fellowship, and 4) to have a minimum of 50% of the graduates of this program pursue a career in Allergy and Immunology research. There are generally 6-8 Allergy/immunology fellows in the UW program, and 4 fellows supported by the WISCART program. All fellows are MDs who have completed residency training in either Pediatrics or Internal Medicine. The first year of the program is funded by monies provided by the departmental and divisional funds, and is primarily clinical in nature. After the clinical year, the second and third years (2 positions per year), which are research intensive, are funded by WISCART. Additional years of training are encouraged, and are funded by research grants or other sources. The WISCART trainers are a diverse group of clinical, translational and basic scientists, and there are an exceptional number of opportunities for trainee research projects. The curriculum takes advantage of a number of institutional resources, including the University of Wisconsin Institute of Clinical and Translational Research, to provide comprehensive clinical research training, and optional advanced degree programs in clinical research.

Public Health Relevance

(See Instructions): Allergic diseases and asthma are among the most common diseases, and are increasing for reasons that are not well understood. The goal of this program is to provide training in clinical and translational research to physician scientists who wish to pursue careers devoted to research in these critical areas of unmet medical need.

Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Institutional National Research Service Award (T32)
Project #
5T32AI007635-13
Application #
8319566
Study Section
Allergy & Clinical Immunology-1 (AITC)
Program Officer
Prograis, Lawrence J
Project Start
2000-08-01
Project End
2015-08-31
Budget Start
2012-09-01
Budget End
2013-08-31
Support Year
13
Fiscal Year
2012
Total Cost
$217,693
Indirect Cost
$18,945
Name
University of Wisconsin Madison
Department
Pediatrics
Type
Schools of Medicine
DUNS #
161202122
City
Madison
State
WI
Country
United States
Zip Code
53715
Esquivel, Ann; Busse, William W (2016) Anaphylaxis Conundrum: A Trojan Horse Phenomenon. J Allergy Clin Immunol Pract :
Kakumanu, Sujani; Jaffee, Katy; Visness, Cynthia M et al. (2016) The influence of atopy and asthma on immune responses in inner-city adults. Immun Inflamm Dis 4:80-90
Sterkel, Alana K; Lorenzini, Jenna L; Fites, J Scott et al. (2016) Fungal Mimicry of a Mammalian Aminopeptidase Disables Innate Immunity and Promotes Pathogenicity. Cell Host Microbe 19:361-74
Branchfield, Kelsey; Nantie, Leah; Verheyden, Jamie M et al. (2016) Pulmonary neuroendocrine cells function as airway sensors to control lung immune response. Science 351:707-10
Bell, Matthew C; Busse, William W (2015) Is It Asthma or Is It COPD: The Overlap Syndrome. J Allergy Clin Immunol Pract 3:641-2; quiz 643
Thomas, Amy O; Jackson, Daniel J; Evans, Michael D et al. (2015) Sex-related differences in pulmonary physiologic outcome measures in a high-risk birth cohort. J Allergy Clin Immunol 136:282-7
Coleman, Amaziah T; Jackson, Daniel J; Gangnon, Ronald E et al. (2015) Comparison of risk factors for viral and nonviral asthma exacerbations. J Allergy Clin Immunol 136:1127-9.e4
Gerald, Joe K; Gerald, Lynn B; Vasquez, Monica M et al. (2015) Markers of Differential Response to Inhaled Corticosteroid Treatment Among Children with Mild Persistent Asthma. J Allergy Clin Immunol Pract 3:540-6.e3
Nanjappa, Som Gowda; Hernández-Santos, Nydiaris; Galles, Kevin et al. (2015) Intrinsic MyD88-Akt1-mTOR Signaling Coordinates Disparate Tc17 and Tc1 Responses during Vaccine Immunity against Fungal Pneumonia. PLoS Pathog 11:e1005161
Starnes, Taylor W; Bennin, David A; Bing, Xinyu et al. (2014) The F-BAR protein PSTPIP1 controls extracellular matrix degradation and filopodia formation in macrophages. Blood 123:2703-14

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