The function of this Administration Core is to provide the UCACE Director with the organizational, administrative, and secretarial support to enable the scientific and clinical research goals of the program to be accomplished. The UCACE Office will be located within the ample office space allocated to the Section of Rheumatology, adjacent to the laboratories of Drs. Clark, Wilson and Weigert. Specific responsibilities of this core, embodied within the UCACE Office are as follows: 1. Coordinate the scientific activities of the program 2. Coordinate the clinical trials program 3. Oversee the documentation related to the clinical trials program 4. Facilitate interactions between investigators at the formal and informal level 5. Coordinate a standing External Scientific Advisory Committee. 6. Prepare annual progress reports 7. Organize annual formal review 8. Organize visits of other external scientists and consultants 9. Provide day to day administrative (fiscal and secretarial) support to investigators 10. Organize and administer monthly UCACE Research Seminars 11. Keep accurate records and accounts on resource utilization 12. Arrange for yearly travel of Director and Project/Core Leaders to national meetings Support services will be provided equally to all Projects and Cores.

Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Program--Cooperative Agreements (U19)
Project #
5U19AI082724-04
Application #
8378422
Study Section
Special Emphasis Panel (ZAI1-QV-I)
Project Start
Project End
Budget Start
2012-06-01
Budget End
2013-05-31
Support Year
4
Fiscal Year
2012
Total Cost
$61,491
Indirect Cost
$22,074
Name
University of Chicago
Department
Type
DUNS #
005421136
City
Chicago
State
IL
Country
United States
Zip Code
60637
Henault, Jill; Riggs, Jeffrey M; Karnell, Jodi L et al. (2016) Self-reactive IgE exacerbates interferon responses associated with autoimmunity. Nat Immunol 17:196-203
Ko, Kichul; Wang, Jianing; Perper, Stuart et al. (2016) Bcl-2 as a Therapeutic Target in Human Tubulointerstitial Inflammation. Arthritis Rheumatol 68:2740-2751
Ho, Irvin Y; Bunker, Jeffrey J; Erickson, Steven A et al. (2016) Refined protocol for generating monoclonal antibodies from single human and murine B cells. J Immunol Methods 438:67-70
Neu, Karlynn E; Henry Dunand, Carole J; Wilson, Patrick C (2016) Heads, stalks and everything else: how can antibodies eradicate influenza as a human disease? Curr Opin Immunol 42:48-55
Trotter, Kimberly; Clark, Marcus R; Liarski, Vladimir M (2016) Overview of pathophysiology and treatment of human lupus nephritis. Curr Opin Rheumatol 28:460-7
DiLillo, David J; Palese, Peter; Wilson, Patrick C et al. (2016) Broadly neutralizing anti-influenza antibodies require Fc receptor engagement for in vivo protection. J Clin Invest 126:605-10
Henry Dunand, Carole J; Leon, Paul E; Huang, Min et al. (2016) Both Neutralizing and Non-Neutralizing Human H7N9 Influenza Vaccine-Induced Monoclonal Antibodies Confer Protection. Cell Host Microbe 19:800-13
Victora, Gabriel D; Wilson, Patrick C (2015) Germinal center selection and the antibody response to influenza. Cell 163:545-8
Cobey, Sarah; Wilson, Patrick; Matsen 4th, Frederick A (2015) The evolution within us. Philos Trans R Soc Lond B Biol Sci 370:
Smith, Mia J; Packard, Thomas A; O'Neill, Shannon K et al. (2015) Loss of anergic B cells in prediabetic and new-onset type 1 diabetic patients. Diabetes 64:1703-12

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