This subproject is one of many research subprojects utilizing theresources provided by a Center grant funded by NIH/NCRR. The subproject andinvestigator (PI) may have received primary funding from another NIH source,and thus could be represented in other CRISP entries. The institution listed isfor the Center, which is not necessarily the institution for the investigator.Hypothesis: polymorphisms in cellular gene products that interact with virus proteins will modulate control of viral replication.cleeular resistance may vary based on drug transporter genetic polymorphinsms.
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