This subproject is one of many research subprojects utilizing theresources provided by a Center grant funded by NIH/NCRR. The subproject andinvestigator (PI) may have received primary funding from another NIH source,and thus could be represented in other CRISP entries. The institution listed isfor the Center, which is not necessarily the institution for the investigator.The central hypothesis of the application is that the LBM loss observed in HNC survivors is primarily a process of cachexia (i.e., increased muscle degradation), not anorexia (i.e., decreased nutrient intake). Hence, a combined intervention of tailored low-intensity resistance training with specialized nutrition therapy is more likely to attenuate the loss of LBM in HNC patients during and after CCR treatment.
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