This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. In our recently completed intervention study commencing in 1999, we have been examining the mechanisms of physical activity behavior by tracking adolescent females for a full school year in a controlled exercise intervention. This intervention was unique in several ways: 1) it targeted sedentary, unfit adolescent females; 2) it tracked not only behavior and fitness over time but also likely psychosocial mediators of behavior change (e.g., self-efficacy, social support, perceived barriers to activity, etc ); and 3) it incorporated both psychosocial and physiological hypotheses in an integrated study with a physiological endpoint. Results demonstrate that the intervention was successful in terms of enhancing physical fitness and physical activity levels among sedentary adolescent females; yet, none of the proposed psychosocial mediators assessed were able to explain the behavior change, and the impact of the intervention on participants' fitness (i.e., VO2max, percent body fat) was minimal. What these findings suggest is that we need to move in new directions in order to identify the salient factors shaping adolescent physical activity behavior and use this knowledge to design more effective interventions for promoting increased activity. That is, we need to gain a better understanding of the natural process by which adolescents become increasingly sedentary with maturation, so that we can do a better job of reversing this process. The currently proposed study extends our findings to date by testing a novel biobehavioral model drawn from recent theoretical and empirical work. This work focuses on exercise-related affect (both acute and enduring) as a dominant factor shaping adolescent physical activity behavior . The role of exercise-associated affect as a factor shaping levels of physical activity participation is an area that has been seriously understudied. Two distinct types of affect have been proposed to contribute to individual behavior choices in terms of physical activity participation: acute exercise-induced affect, and global or enduring exercise-associated affect. The first refers to the transient, immediate mood effects that occur following a single exercise bout, whereas the second refers to an individuals' more pervasive, integrated perception of how much s/he enjoys exercising. Both types of exercise-associated affect will be explored in this proposal, as described in the following hypotheses.

Agency
National Institute of Health (NIH)
Institute
National Center for Research Resources (NCRR)
Type
General Clinical Research Centers Program (M01)
Project #
2M01RR000827-31
Application #
7374305
Study Section
National Center for Research Resources Initial Review Group (RIRG)
Project Start
2006-04-01
Project End
2006-11-30
Budget Start
2006-04-01
Budget End
2006-11-30
Support Year
31
Fiscal Year
2006
Total Cost
$56,235
Indirect Cost
Name
University of California San Diego
Department
Type
Schools of Medicine
DUNS #
804355790
City
La Jolla
State
CA
Country
United States
Zip Code
92093
Grams, Morgan E; Sang, Yingying; Ballew, Shoshana H et al. (2018) Predicting timing of clinical outcomes in patients with chronic kidney disease and severely decreased glomerular filtration rate. Kidney Int 93:1442-1451
Lavigne, Katie M; Woodward, Todd S (2018) Hallucination- and speech-specific hypercoupling in frontotemporal auditory and language networks in schizophrenia using combined task-based fMRI data: An fBIRN study. Hum Brain Mapp 39:1582-1595
Milot, Marie-Hélène; Marchal-Crespo, Laura; Beaulieu, Louis-David et al. (2018) Neural circuits activated by error amplification and haptic guidance training techniques during performance of a timing-based motor task by healthy individuals. Exp Brain Res 236:3085-3099
Hsu, Simon; Rifkin, Dena E; Criqui, Michael H et al. (2018) Relationship of femoral artery ultrasound measures of atherosclerosis with chronic kidney disease. J Vasc Surg 67:1855-1863.e1
Inker, Lesley A; Grams, Morgan E; Levey, Andrew S et al. (2018) Relationship of Estimated GFR and Albuminuria to Concurrent Laboratory Abnormalities: An Individual Participant Data Meta-analysis in a Global Consortium. Am J Kidney Dis :
Egnot, Natalie Suder; Barinas-Mitchell, Emma; Criqui, Michael H et al. (2018) An exploratory factor analysis of inflammatory and coagulation markers associated with femoral artery atherosclerosis in the San Diego Population Study. Thromb Res 164:9-14
Juraschek, Stephen P; Miller 3rd, Edgar R; Appel, Lawrence J (2018) Orthostatic Hypotension and Symptoms in the AASK Trial. Am J Hypertens 31:665-671
Chen, Teresa K; Appel, Lawrence J; Grams, Morgan E et al. (2017) APOL1 Risk Variants and Cardiovascular Disease: Results From the AASK (African American Study of Kidney Disease and Hypertension). Arterioscler Thromb Vasc Biol 37:1765-1769
Juraschek, Stephen P; Appel, Lawrence J; Miller 3rd, Edgar R (2017) Metoprolol Increases Uric Acid and Risk of Gout in African Americans With Chronic Kidney Disease Attributed to Hypertension. Am J Hypertens 30:871-875
Chen, Teresa K; Tin, Adrienne; Peralta, Carmen A et al. (2017) APOL1 Risk Variants, Incident Proteinuria, and Subsequent eGFR Decline in Blacks with Hypertension-Attributed CKD. Clin J Am Soc Nephrol 12:1771-1777

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