The PI will serve as the Director of the Administrative Core. The current Administrative Core of CCRTD iscomposed of a scientific director, manager of administrative services and an administrative secretary. Itserves as an intermediate between the cancer researchers and the bureaucracy of the University. The mainobjectives of the Administrative Core are a) to provide leadership and focal point for organization of allactivities of the proposed Center of Excellence; b) to serve as a medium between the components of theproposed Center of Excellence and NCMHD; and c) to provide financial management, purchasing and otheradministrative services to the faculty and staff of the proposed Center of Excellence. We will utilize currentadvisory committees to oversee the activities of the new center. These are the Institutional AdvisoryCommittee (IAC), the External Advisory Committee (EAC), and the Local External Advisory Committee(LEAC). The Core Director will oversee all day-to-day administrative responsibilities of proposed centerwhich include implementing the policies of the program after consultation and guidance from the IAC, EACand the LEAC. The Administrative Core is designed to maintain a strong and seamless communication withmembers of CCRTD and their research staff. The administrative staff provides scientists with the services ofpost-award management duties such as establishment of University budget accounts for each grant,processing purchase requisitions for each grant, interacting with the staffs of the Offices of Grants andContracts, Research and Sponsored Programs, and Purchasing and Accounts Payable and preparingmonthly budget printouts for each scientist. The Administrative Core also organizes weekly group meetings,weekly invited speaker series and annual symposia. The Administrative Core will coordinate the activities ofall components and cores of the proposed Center of Excellence.

Agency
National Institute of Health (NIH)
Institute
National Institute on Minority Health and Health Disparities (NIMHD)
Type
Exploratory Grants (P20)
Project #
1P20MD002285-01
Application #
7304959
Study Section
Special Emphasis Panel (ZRG1-DIG-C (52))
Project Start
2007-10-01
Project End
2012-05-30
Budget Start
2007-10-01
Budget End
2008-05-31
Support Year
1
Fiscal Year
2007
Total Cost
$168,145
Indirect Cost
Name
Clark Atlanta University
Department
Type
DUNS #
065325177
City
Atlanta
State
GA
Country
United States
Zip Code
30314
Elliott, Bethtrice; Zackery, DeAdra L; Eaton, Vanessa A et al. (2018) Ethnic differences in TGF?-signaling pathway may contribute to prostate cancer health disparity. Carcinogenesis 39:546-555
Kimbrough-Allah, Mawiyah N; Millena, Ana C; Khan, Shafiq A (2018) Differential role of PTEN in transforming growth factor ? (TGF-?) effects on proliferation and migration in prostate cancer cells. Prostate 78:377-389
Scarlett, Kisha A; White, El-Shaddai Z; Coke, Christopher J et al. (2018) Agonist-induced CXCR4 and CB2 Heterodimerization Inhibits G?13/RhoA-mediated Migration. Mol Cancer Res 16:728-739
Caggia, Silvia; Chunduri, HimaBindu; Millena, Ana C et al. (2018) Novel role of Gi?2 in cell migration: Downstream of PI3-kinase-AKT and Rac1 in prostate cancer cells. J Cell Physiol 234:802-815
Hawsawi, Ohuod; Henderson, Veronica; Burton, Liza J et al. (2018) High mobility group A2 (HMGA2) promotes EMT via MAPK pathway in prostate cancer. Biochem Biophys Res Commun 504:196-202
Burton, Liza J; Henderson, Veronica; Liburd, Latiffa et al. (2017) Snail transcription factor NLS and importin ?1 regulate the subcellular localization of Cathepsin L and Cux1. Biochem Biophys Res Commun 491:59-64
Morton, Derrick J; Patel, Divya; Joshi, Jugal et al. (2017) ID4 regulates transcriptional activity of wild type and mutant p53 via K373 acetylation. Oncotarget 8:2536-2549
Barrett, Cachétne S X; Millena, Ana C; Khan, Shafiq A (2017) TGF-? Effects on Prostate Cancer Cell Migration and Invasion Require FosB. Prostate 77:72-81
Joshi, Jugal Bharat; Patel, Divya; Morton, Derrick J et al. (2017) Inactivation of ID4 promotes a CRPC phenotype with constitutive AR activation through FKBP52. Mol Oncol 11:337-357
Yu, Min; Wang, Ulrica; Wang, Zhengxin (2016) E2F and GATA switches turn off WD repeat domain 77 expression in differentiating cells. Biochem J 473:2331-43

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