The Cancer Prevention and Control Program (CPCP) is a multidisciplinary research effort, using intervention techniques, to reduce the overall and specific cancer incidence, morbidity, and mortality in men and women across the age spectrum through research aimed at: 1) identifying effective lifestyle (e.g. tobacco use, diet, exercise, and weight control) and medical (e.g. chemoprevention, vaccinations) interventions to reduce the cancer burden;2) understanding environmental determinants of lifestyles, behaviors and health outcomes and implementing community interventions to modify these environmental determinants;3) determining optimal early diagnostic techniques and detection programs and their utilization;and 4) conducting and evaluating interventions designed to improve the quality of cancer care and management, i.e. survivorship. Cross cutting these research areas is a focus on understanding and reducing the unequal burden of cancer in specific population groups. These goals are obtainable by combining the expertise and resources of Stanford University (SU), in particular the Stanford Prevention Research Center (SPRC), Clinical Cancer Genetics Center, Stanford Center on Stress and Health, Cancer Center Shared Resources, and the Northern California Cancer Center (NCCC). Extensive networks with community organizations and physician groups, including many that work with underserved populations, strengthen the CPCP's research program. The Cancer Prevention and Control Program includes 26 members from 10 SU Departments, including 3 Schools, and the NCCC. Plans for the next three years include: 1) enhancing intra-programmatic collaborations among program members;2) identifying translational opportunities and building strong inter-programmatic collaborations, particularly with investigators in the Cancer Epidemiology Program (Program 9), but also with other Cancer Center Programs and the Shared Resources;3) tailoring current SPRC lifestyle intervention and assessment research to populations at greatest risk for specific types of cancer;and 4) strengthening the health policy research agenda for cancer prevention.

Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Center Core Grants (P30)
Project #
5P30CA124435-03
Application #
7826895
Study Section
Subcommittee G - Education (NCI)
Project Start
Project End
Budget Start
2009-06-01
Budget End
2010-05-31
Support Year
3
Fiscal Year
2009
Total Cost
$20,912
Indirect Cost
Name
Stanford University
Department
Type
DUNS #
009214214
City
Stanford
State
CA
Country
United States
Zip Code
94305
Thorsson, Vésteinn; Gibbs, David L; Brown, Scott D et al. (2018) The Immune Landscape of Cancer. Immunity 48:812-830.e14
Rogers, Zoë N; McFarland, Christopher D; Winters, Ian P et al. (2018) Mapping the in vivo fitness landscape of lung adenocarcinoma tumor suppression in mice. Nat Genet 50:483-486
Nair, Viswam S; Sundaram, Vandana; Desai, Manisha et al. (2018) Accuracy of Models to Identify Lung Nodule Cancer Risk in the National Lung Screening Trial. Am J Respir Crit Care Med 197:1220-1223
She, Richard; Jarosz, Daniel F (2018) Mapping Causal Variants with Single-Nucleotide Resolution Reveals Biochemical Drivers of Phenotypic Change. Cell 172:478-490.e15
Champion, Magali; Brennan, Kevin; Croonenborghs, Tom et al. (2018) Module Analysis Captures Pancancer Genetically and Epigenetically Deregulated Cancer Driver Genes for Smoking and Antiviral Response. EBioMedicine 27:156-166
Zhou, Mu; Leung, Ann; Echegaray, Sebastian et al. (2018) Non-Small Cell Lung Cancer Radiogenomics Map Identifies Relationships between Molecular and Imaging Phenotypes with Prognostic Implications. Radiology 286:307-315
Pollom, Erqi L; Fujimoto, Dylann K; Han, Summer S et al. (2018) Newly diagnosed glioblastoma: adverse socioeconomic factors correlate with delay in radiotherapy initiation and worse overall survival. J Radiat Res 59:i11-i18
Nørgaard, Caroline Holm; Jakobsen, Lasse Hjort; Gentles, Andrew J et al. (2018) Subtype assignment of CLL based on B-cell subset associated gene signatures from normal bone marrow - A proof of concept study. PLoS One 13:e0193249
Im, Hogune; Rao, Varsha; Sridhar, Kunju et al. (2018) Distinct transcriptomic and exomic abnormalities within myelodysplastic syndrome marrow cells. Leuk Lymphoma 59:2952-2962
Huang, Min; Zhu, Li; Garcia, Jacqueline S et al. (2018) Brd4 regulates the expression of essential autophagy genes and Keap1 in AML cells. Oncotarget 9:11665-11676

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