INTEGRATED HEALTH SCIENCES FACILITY CORE The mission of CEED is to improve human health through translational research using emerging science, engineering, and technology to determine how the environment, genome and epigenome interact to modulate the risk of adverse health outcomes. The Integrated Health Sciences Facility Core (IHSFC) plays a central role in achieving this research mission of the CEED as the nexus for the Center?s interdisciplinary and translational research. The IHSFC facilitates dialog among CEED members from different disciplines and provides guidance and training in design and implementation of studies in population (observational) and clinical epidemiology. It provides expertise in a wide range of methodologies including controlled exposure, survey research, controlled trials, and clinical panel studies of novel biomarkers. The IHSFC is also the portal for biostatistics, data management, archiving, and security. It also provides expertise and support for human subjects protection including IRB and HIPAA compliance. The Core synergizes with our Community Engagement Core (CoEC), through overlapping faculty and staff, to support participatory research and identify opportunities for recruitment by the IHSFC.

Agency
National Institute of Health (NIH)
Institute
National Institute of Environmental Health Sciences (NIEHS)
Type
Center Core Grants (P30)
Project #
5P30ES005022-33
Application #
9927640
Study Section
Environmental Health Sciences Review Committee (EHS)
Project Start
Project End
Budget Start
2020-04-01
Budget End
2021-03-31
Support Year
33
Fiscal Year
2020
Total Cost
Indirect Cost
Name
Rbhs-School of Public Health
Department
Type
DUNS #
078795880
City
Piscataway
State
NJ
Country
United States
Zip Code
08854
Lee, Heedoo; Zhang, Duo; Laskin, Debra L et al. (2018) Functional Evidence of Pulmonary Extracellular Vesicles in Infectious and Noninfectious Lung Inflammation. J Immunol 201:1500-1509
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Barrett, E S; Hoeger, K M; Sathyanarayana, S et al. (2018) Anogenital distance in newborn daughters of women with polycystic ovary syndrome indicates fetal testosterone exposure. J Dev Orig Health Dis 9:307-314
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Tiethof, Angela K; Richardson, Jason R; Hart, Ronald P (2018) Knockdown of Butyrylcholinesterase but Not Inhibition by Chlorpyrifos Alters Early Differentiation Mechanisms in Human Neural Stem Cells. Toxics 6:
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Jabbar, Shaima; Reuhl, Kenneth; Sarkar, Dipak K (2018) Prenatal alcohol exposure increases the susceptibility to develop aggressive prolactinomas in the pituitary gland. Sci Rep 8:7720
Radbel, Jared; Le-Hoang, Oanh; Vayas, Kinal N et al. (2018) Effect of World Trade Center Dust Exposure and Chronic Intermittent Hypoxia on Macrophage Matrix Metalloproteinase-12 Expression in Mice. Ann Am Thorac Soc 15:S125-S126

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