The UNC SBRP will offer traineeships to approximately 6 students per year. The trainees will participate in a laboratory rotation program to build knowledge and skills across disciplines, and be involved in a team project to provide in death information and presentations on a superfund related topic. All the students associated with the UNC-SBRP will participate in a annual Research Symposium to build across project integration and cooperation, as well as learn and practice communication skills. They will also be part of a field experience to learn about how superfund issues are managed and the results of research used. Together, these activities leads to strong teams with knowledge and experiences across the broad of research areas that are part of the UNC-SBRP.

Agency
National Institute of Health (NIH)
Institute
National Institute of Environmental Health Sciences (NIEHS)
Type
Hazardous Substances Basic Research Grants Program (NIEHS) (P42)
Project #
3P42ES005948-11S1
Application #
6587629
Study Section
Special Emphasis Panel (ZES1)
Project Start
2002-04-01
Project End
2003-03-31
Budget Start
Budget End
Support Year
11
Fiscal Year
2002
Total Cost
$175,238
Indirect Cost
Name
University of North Carolina Chapel Hill
Department
Type
DUNS #
078861598
City
Chapel Hill
State
NC
Country
United States
Zip Code
27599
Luo, Yu-Syuan; Hsieh, Nan-Hung; Soldatow, Valerie Y et al. (2018) Comparative analysis of metabolism of trichloroethylene and tetrachloroethylene among mouse tissues and strains. Toxicology 409:33-43
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Luo, Yu-Syuan; Furuya, Shinji; Soldatov, Valerie Y et al. (2018) Metabolism and Toxicity of Trichloroethylene and Tetrachloroethylene in Cytochrome P450 2E1 Knockout and Humanized Transgenic Mice. Toxicol Sci 164:489-500
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Li, Gen; Jima, Dereje; Wright, Fred A et al. (2018) HT-eQTL: integrative expression quantitative trait loci analysis in a large number of human tissues. BMC Bioinformatics 19:95
Adebambo, Oluwadamilare A; Shea, Damian; Fry, Rebecca C (2018) Cadmium disrupts signaling of the hypoxia-inducible (HIF) and transforming growth factor (TGF-?) pathways in placental JEG-3 trophoblast cells via reactive oxygen species. Toxicol Appl Pharmacol 342:108-115
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