Abstract

Communication and organization are key elements in the success of a Research Center Grant. OurAdministrative Core will serve as the communication nexus and provide the organizational structurenecessary for optimal function of the various collaborative groups of this Center Grant. The AdministrativeCore staff will 1) provide efficient and timely dissemination of information to Project and Core leaders, 2)serve as a central repository for grant-related and research-related materials, 3) provide biostatisticalSupport, including expertise in study design and statistical analysis, 4) facilitate the generation of reports andother documents required by NIH in a timely fashion, 5) arrange key conferences and meetings,6) communicate with the Local Advisory Committee and the External Advisory Panel, and 7) serve as theWeb Master for the Center Grant web page on the expanded Department of Surgery's website.

  Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Specialized Center (P50)
Project #
1P50GM069790-01A2
Application #
7074166
Study Section
Special Emphasis Panel (ZGM1-PPBC-5 (TB))
Project Start
2006-07-01
Project End
2011-05-31
Budget Start
2006-07-01
Budget End
2007-05-31
Support Year
1
Fiscal Year
2006
Total Cost
$122,486
Indirect Cost

  Institution

Name
University of Medicine & Dentistry of NJ
Department
Type
DUNS #
623946217
City
Newark
State
NJ
Country
United States
Zip Code
07107

  Publications

Reino, Diego C; Palange, David; Feketeova, Elenora et al. (2012) Activation of toll-like receptor 4 is necessary for trauma hemorrhagic shock-induced gut injury and polymorphonuclear neutrophil priming. Shock 38:107-14
Kannan, Kolenkode B; Colorado, Iriana; Reino, Diego et al. (2011) Hypoxia-inducible factor plays a gut-injurious role in intestinal ischemia reperfusion injury. Am J Physiol Gastrointest Liver Physiol 300:G853-61
Condon, Michael; Senthil, Maheswari; Xu, Da-Zhong et al. (2011) Intravenous injection of mesenteric lymph produced during hemorrhagic shock decreases RBC deformability in the rat. J Trauma 70:489-95
Qin, Yong; Prescott, Lauriston M; Deitch, Edwin A et al. (2011) Heparin use in a rat hemorrhagic shock model induces biologic activity in mesenteric lymph separate from shock. Shock 35:411-21
Sheth, Sharvil U; Palange, David; Xu, Da-Zhong et al. (2011) Testosterone depletion or blockade in male rats protects against trauma hemorrhagic shock-induced distant organ injury by limiting gut injury and subsequent production of biologically active mesenteric lymph. J Trauma 71:1652-8
Reino, Diego C; Pisarenko, Vadim; Palange, David et al. (2011) Trauma hemorrhagic shock-induced lung injury involves a gut-lymph-induced TLR4 pathway in mice. PLoS One 6:e14829
Sharpe, Susan M; Qin, Xiaofa; Lu, Qi et al. (2010) Loss of the intestinal mucus layer in the normal rat causes gut injury but not toxic mesenteric lymph nor lung injury. Shock 34:475-81
Doucet, Danielle; Badami, Chirag; Palange, David et al. (2010) Estrogen receptor hormone agonists limit trauma hemorrhage shock-induced gut and lung injury in rats. PLoS One 5:e9421
Doucet, Danielle R; Bonitz, R Paul; Feinman, Rena et al. (2010) Estrogenic hormone modulation abrogates changes in red blood cell deformability and neutrophil activation in trauma hemorrhagic shock. J Trauma 68:35-41
Mohr, Alicia M; Lavery, Robert F; Sifri, Ziad C et al. (2010) Gender differences in glucose variability after severe trauma. Am Surg 76:896-902

Showing the most recent 10 out of 23 publications