The Diabetes Research and Training Center (DRTC) at Vanderbilt is one of a network of DRTC's established by the National Institute of Arthritis, Metabolism and Digestive Diseases (NIAMDD) to conduct research and programs for training in diabetes mellitus and related endocrine and metabolic disorders consistent with the National Diabetes Research and Education Act (PL 93-354), with applicable recommendations made by the National Commission on Diabetes, and with the DRTC guidelines promulgated by NIAMDD. Specifically, each DRTC is charged to conduct: a) research in the diagnosis and treatment of diabetes mellitus and related endocrine and metabolic disorders and the complications resulting from such disease or disorders, b) training programs for physicians and allied health personnel in current methods of diagnosis and treatment of such disease, disorders, and complications, and c) information programs for physicians and allied health personnel who provide primary care for patients with such disease, disorders, or complications. Although the Centers necessarily have the same general objectives, the Vanderbilt DRTC, like every individual DRTC, has been set up in a manner that best meets the special needs of its own region. The Vanderbilt DRTC consists of two coordinated divisions: Basic Science Division that includes most of the diabetes research and research training programs that were initiated at Vanderbilt under the egies of the Diabetes-Endocrinology Research Center (1973-1978), and a Clinical Science Division that is responsible for the educational, research, training, evaluation, and outreach activities embodied in the DRTC concept.

Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
Comprehensive Center (P60)
Project #
5P60DK020593-08
Application #
3108389
Study Section
(SRC)
Project Start
1978-09-01
Project End
1989-11-30
Budget Start
1985-12-01
Budget End
1986-11-30
Support Year
8
Fiscal Year
1986
Total Cost
Indirect Cost
Name
Vanderbilt University Medical Center
Department
Type
Schools of Medicine
DUNS #
004413456
City
Nashville
State
TN
Country
United States
Zip Code
37203
Dutter, Brendan F; Ender, Anna; Sulikowski, Gary A et al. (2018) Rhodol-based thallium sensors for cellular imaging of potassium channel activity. Org Biomol Chem 16:5575-5579
Herrick, Mary K; Favela, Kristin M; Simerly, Richard B et al. (2018) Attenuation of diet-induced hypothalamic inflammation following bariatric surgery in female mice. Mol Med 24:56
Perez, Katia M; Curley, Kathleen L; Slaughter, James C et al. (2018) Glucose Homeostasis and Energy Balance in Children With Pseudohypoparathyroidism. J Clin Endocrinol Metab 103:4265-4274
Marre, Meghan L; McGinty, John W; Chow, I-Ting et al. (2018) Modifying Enzymes Are Elicited by ER Stress, Generating Epitopes That Are Selectively Recognized by CD4+ T Cells in Patients With Type 1 Diabetes. Diabetes 67:1356-1368
Creecy, Amy; Uppuganti, Sasidhar; Unal, Mustafa et al. (2018) Low bone toughness in the TallyHO model of juvenile type 2 diabetes does not worsen with age. Bone 110:204-214
Russart, Kathryn L G; Huk, Danielle; Nelson, Randy J et al. (2018) Elevated aggressive behavior in male mice with thyroid-specific Prkar1a and global Epac1 gene deletion. Horm Behav 98:121-129
Coppola, Jennifer J; Disney, Anita A (2018) Most calbindin-immunoreactive neurons, but few calretinin-immunoreactive neurons, express the m1 acetylcholine receptor in the middle temporal visual area of the macaque monkey. Brain Behav 8:e01071
Zhu, Lin; Luu, Thao; Emfinger, Christopher H et al. (2018) CETP Inhibition Improves HDL Function but Leads to Fatty Liver and Insulin Resistance in CETP-Expressing Transgenic Mice on a High-Fat Diet. Diabetes 67:2494-2506
Lu, Sichang; McGough, Madison A P; Shiels, Stefanie M et al. (2018) Settable polymer/ceramic composite bone grafts stabilize weight-bearing tibial plateau slot defects and integrate with host bone in an ovine model. Biomaterials 179:29-45
Horwitz, Elad; Krogvold, Lars; Zhitomirsky, Sophia et al. (2018) ?-Cell DNA Damage Response Promotes Islet Inflammation in Type 1 Diabetes. Diabetes 67:2305-2318

Showing the most recent 10 out of 1487 publications