|
Sang, Tzu-Kang; Li, Chenjian; Liu, Wencheng et al. (2005) Inactivation of Drosophila Apaf-1 related killer suppresses formation of polyglutamine aggregates and blocks polyglutamine pathogenesis. Hum Mol Genet 14:357-72
|
|
Chen, P; Ho, S-I; Shi, Z et al. (2004) Bifunctional killing activity encoded by conserved reaper proteins. Cell Death Differ 11:704-13
|
|
Salvesen, Guy S; Abrams, John M (2004) Caspase activation - stepping on the gas or releasing the brakes? Lessons from humans and flies. Oncogene 23:2774-84
|
|
Kauppila, Saila; Maaty, Walid S A; Chen, Po et al. (2003) Eiger and its receptor, Wengen, comprise a TNF-like system in Drosophila. Oncogene 22:4860-7
|
|
Sogame, Naoko; Kim, Misoo; Abrams, John M (2003) Drosophila p53 preserves genomic stability by regulating cell death. Proc Natl Acad Sci U S A 100:4696-701
|
|
Rodriguez, Antony; Chen, Po; Oliver, Holt et al. (2002) Unrestrained caspase-dependent cell death caused by loss of Diap1 function requires the Drosophila Apaf-1 homolog, Dark. EMBO J 21:2189-97
|
|
Christich, Anna; Kauppila, Saila; Chen, Po et al. (2002) The damage-responsive Drosophila gene sickle encodes a novel IAP binding protein similar to but distinct from reaper, grim, and hid. Curr Biol 12:137-40
|
|
Corey, D R; Abrams, J M (2001) Morpholino antisense oligonucleotides: tools for investigating vertebrate development. Genome Biol 2:REVIEWS1015
|
|
Brodsky, M H; Nordstrom, W; Tsang, G et al. (2000) Drosophila p53 binds a damage response element at the reaper locus. Cell 101:103-13
|
|
Chen, P; Abrams, J M (2000) Analysis of programmed cell death and apoptosis in Drosophila. Methods Enzymol 322:65-76
|
Showing the most recent 10 out of 18 publications
