Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Project (R01)
Project #
2R01AI010947-15
Application #
3960673
Study Section
Project Start
Project End
Budget Start
Budget End
Support Year
15
Fiscal Year
1986
Total Cost
Indirect Cost
Name
Vanderbilt University Medical Center
Department
Type
DUNS #
004413456
City
Nashville
State
TN
Country
United States
Zip Code
37203
Chan, John; Mehta, Simren; Bharrhan, Sushma et al. (2014) The role of B cells and humoral immunity in Mycobacterium tuberculosis infection. Semin Immunol 26:588-600
Kupershmidt, S; Rall, G F; Lu, Z Q et al. (1992) Cleavage of concatemeric DNA at the internal junction of ""translocation"" mutants of pseudorabies virus and inversion of their L component appear to be linked. Virology 187:223-32
Rall, G F; Kupershmidt, S; Sugg, N et al. (1992) Functions of the sequences at the ends of the inverted repeats of pseudorabies virus. J Virol 66:1506-19
Zsak, L; Sugg, N; Ben-Porat, T (1992) The different interactions of a gIII mutant of pseudorabies virus with several different cell types. J Gen Virol 73 ( Pt 4):821-7
Zsak, L; Zuckermann, F; Sugg, N et al. (1992) Glycoprotein gI of pseudorabies virus promotes cell fusion and virus spread via direct cell-to-cell transmission. J Virol 66:2316-25
Reilly, L M; Rall, G; Lomniczi, B et al. (1991) The ability of pseudorabies virus to grow in different hosts is affected by the duplication and translocation of sequences from the left end of the genome to the UL-US junction. J Virol 65:5839-47
Rall, G F; Lu, Z Q; Sugg, N et al. (1991) Acquisition of an additional internal cleavage site differentially affects the ability of pseudorabies virus to multiply in different host cells. J Virol 65:6604-11
Zsak, L; Sugg, N; Ben-Porat, T et al. (1991) The gIII glycoprotein of pseudorabies virus is involved in two distinct steps of virus attachment. J Virol 65:4317-24
Kupershmidt, S; DeMarchi, J M; Lu, Z Q et al. (1991) Analysis of an origin of DNA replication located at the L terminus of the genome of pseudorabies virus. J Virol 65:6283-91
Rall, G F; Kupershmidt, S; Lu, X Q et al. (1991) Low-level inversion of the L component of pseudorabies virus is not dependent on sequence homology. J Virol 65:7016-9

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