Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Research Project (R01)
Project #
3R01CA032827-03S2
Application #
3170666
Study Section
(SSS)
Project Start
1982-08-01
Project End
1986-04-30
Budget Start
1984-08-01
Budget End
1986-04-30
Support Year
3
Fiscal Year
1986
Total Cost
Indirect Cost
Name
Stanford University
Department
Type
Schools of Medicine
DUNS #
800771545
City
Stanford
State
CA
Country
United States
Zip Code
94305
Ning, S; Hahn, G M (1994) Formation of tight junctions and desmosomes protects MDCK cells against hyperthermic killing. J Cell Physiol 160:249-54
Koong, A C; Giaccia, A J; Hahn, G M et al. (1993) Activation of potassium channels by hypoxia and reoxygenation in the human lung adenocarcinoma cell line A549. J Cell Physiol 156:341-7
Saad, A H; Hahn, G M (1992) Activation of potassium channels: relationship to the heat shock response. Proc Natl Acad Sci U S A 89:9396-9
Anderson, R L; Kraft, P E; Bensaude, O et al. (1991) Binding activity of glucocorticoid receptors after heat shock. Exp Cell Res 197:100-6
Mehdi, S Q; Hahn, G M (1990) Effects of hyperthermic temperatures and the synthesis of heat-shock proteins on the lateral diffusion of H-2Kk. Int J Hyperthermia 6:553-61
Gonzalez-Mendez, R R; Hahn, G M (1989) Effects of hyperthermia on the intracellular pH and membrane potential of Chinese hamster ovary cells. Int J Hyperthermia 5:69-84
Lin, P P; Hahn, G M (1988) Growth factors and hyperthermia. II. Viability of Chinese hamster ovary HA-1 cells during serum starvation and hyperthermia. Radiat Res 113:513-25
Woo, S Y; Rice, G C; Kapp, D S et al. (1988) A predictive assay for human tumor cellular response to hyperthermia using dansyl lysine staining and flow cytometry. Int J Radiat Oncol Biol Phys 14:361-5
Lin, P P; Hahn, G M (1988) Growth factors and hyperthermia. I. The relationship between hyperthermic cell killing and the mitogenic response to serum and growth factors. Radiat Res 113:501-12
Lee, K J; Hahn, G M (1988) Abnormal proteins as the trigger for the induction of stress responses: heat, diamide, and sodium arsenite. J Cell Physiol 136:411-20

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