Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Research Project (R01)
Project #
1R01CA048943-01
Application #
3192831
Study Section
Chemical Pathology Study Section (CPA)
Project Start
1988-12-01
Project End
1991-11-30
Budget Start
1988-12-01
Budget End
1989-11-30
Support Year
1
Fiscal Year
1989
Total Cost
Indirect Cost
Name
Salk Institute for Biological Studies
Department
Type
DUNS #
005436803
City
La Jolla
State
CA
Country
United States
Zip Code
92037
Lee, Ji Shin; Fackler, Mary Jo; Lee, Jae Hyuk et al. (2010) Basal-like breast cancer displays distinct patterns of promoter methylation. Cancer Biol Ther 9:1017-24
Loeb, D M; Summers, J L; Burwell, E A et al. (2003) An isoform of the Wilms' tumor suppressor gene potentiates granulocytic differentiation. Leukemia 17:965-71
Loeb, David M; Korz, Dorian; Katsnelson, Michael et al. (2002) Cyclin E is a target of WT1 transcriptional repression. J Biol Chem 277:19627-32
Ferguson, A T; Evron, E; Umbricht, C B et al. (2000) High frequency of hypermethylation at the 14-3-3 sigma locus leads to gene silencing in breast cancer. Proc Natl Acad Sci U S A 97:6049-54
Carey, L A; Kim, N W; Goodman, S et al. (1999) Telomerase activity and prognosis in primary breast cancers. J Clin Oncol 17:3075-81
Nacht, M; Ferguson, A T; Zhang, W et al. (1999) Combining serial analysis of gene expression and array technologies to identify genes differentially expressed in breast cancer. Cancer Res 59:5464-70
Varon, D; Jiang, C; Hedican, C et al. (1997) Telomerase activity in the normal and neoplastic rat mammary gland. Cancer Res 57:5605-9
McKenzie, K E; Armstrong, B A; Chen, Y et al. (1997) Alterations in the Ha-ras-1 and the p53 pathway genes in the progression of N-methyl-N-nitrosourea-induced rat mammary tumors. Mol Carcinog 20:194-203
Glebov, O K; McKenzie, K E; White, C A et al. (1994) Frequent p53 gene mutations and novel alleles in familial breast cancer. Cancer Res 54:3703-9
Runnebaum, I B; Yee, J K; Kieback, D G et al. (1994) Wild-type p53 suppresses the malignant phenotype in breast cancer cells containing mutant p53 alleles. Anticancer Res 14:1137-44

Showing the most recent 10 out of 20 publications