Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Research Project (R01)
Project #
5R01CA050434-08
Application #
2093771
Study Section
Chemical Pathology Study Section (CPA)
Project Start
1989-08-01
Project End
1998-05-31
Budget Start
1996-06-01
Budget End
1998-05-31
Support Year
8
Fiscal Year
1996
Total Cost
Indirect Cost
Name
New York University
Department
Pathology
Type
Schools of Medicine
DUNS #
City
New York
State
NY
Country
United States
Zip Code
10016
Osei-Sarfo, Kwame; de Castro, Ignacio Perez; Pellicer, Angel (2012) p15(INK4b) plays a crucial role in murine lymphoid development and tumorigenesis. Carcinogenesis 33:708-13
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Perez de Castro, Ignacio; Bivona, Trever G; Philips, Mark R et al. (2004) Ras activation in Jurkat T cells following low-grade stimulation of the T-cell receptor is specific to N-Ras and occurs only on the Golgi apparatus. Mol Cell Biol 24:3485-96
Hernandez-Munoz, Inmaculada; Benet, Marta; Calero, Miguel et al. (2003) rgr oncogene: activation by elimination of translational controls and mislocalization. Cancer Res 63:4188-95
Leonardi, Peter; Kassin, Ezra; Hernandez-Munoz, Inmaculada et al. (2002) Human rgr: transforming activity and alteration in T-cell malignancies. Oncogene 21:5108-16
Zagzag, D; Miller, D C; Knopp, E et al. (2000) Primitive neuroectodermal tumors of the brainstem: investigation of seven cases. Pediatrics 106:1045-53
Hernandez-Munoz, I; Malumbres, M; Leonardi, P et al. (2000) The Rgr oncogene (homologous to RalGDS) induces transformation and gene expression by activating Ras, Ral and Rho mediated pathways. Oncogene 19:2745-57

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